The Art of Manliness - May 08, 2023


Optimize Your Testosterone


Episode Stats

Length

46 minutes

Words per Minute

175.48276

Word Count

8,109

Sentence Count

496

Misogynist Sentences

11

Hate Speech Sentences

8


Summary

In this episode, Dr. Kyle Gillette joins me to discuss testosterone and hormone optimization. Dr. Gillette is a family physician who specializes in hormone optimization, helping people have healthy hormones so they live a flourishing life. In this episode we discuss a quick overview of the different hormones that affect male health and how to optimize your testosterone. We then discuss what causes low testosterone in individual men, and how its decline in the general male population may be linked to both birth control and World War II. Finally, we discuss the risk and benefits of taking testosterone, and what young men can do to prepare for a lifetime of optimal testosterone and hormonal health.


Transcript

00:00:00.000 Hey, this is Brett, we're taking a break from new episodes today, so we're rebroadcasting
00:00:02.840 episode number 893, Optimize Your Testosterone with Dr. Kyle Gillette.
00:00:07.180 Hope you enjoy it.
00:00:07.860 We'll see you on Monday with a brand new episode.
00:00:16.520 Brett McKay here, and welcome to another edition of the Art of Manliness podcast.
00:00:20.640 When men think about optimizing their hormones, they tend only to think about raising their
00:00:24.500 testosterone.
00:00:25.500 But while increasing T can be important, an ideal health profile also means having testosterone
00:00:29.980 that's in balance with your other hormones as well.
00:00:32.700 Today on the show, Dr. Kyle Gillette joins me to discuss both of those prongs of all-around
00:00:36.760 hormone optimization.
00:00:38.400 We start with a quick overview of the different hormones that affect male health.
00:00:42.000 We then get into what qualifies as low testosterone and how to accurately test yours.
00:00:46.480 We also discuss what causes low testosterone in individual men and how its decline in the
00:00:51.020 general male population may be linked to both birth control and the world wars.
00:00:55.520 In the second half of our conversation, we discuss how to both raise testosterone and get
00:00:59.380 rid of excess estrogen, including the use of some effective supplements you may never
00:01:03.460 heard of.
00:01:04.160 We then get into the risk and benefits of taking TRT, before ending our discussion with
00:01:08.520 what young men can do to prepare for a lifetime of optimal T and hormonal health.
00:01:13.100 After the show's over, check out our show notes at awim.is slash optimal T.
00:01:16.420 All right, Dr. Kyle Gillette, welcome to the show.
00:01:35.900 Thank you.
00:01:36.560 My pleasure.
00:01:37.280 So you are a medical doctor.
00:01:39.100 You do family practice.
00:01:40.580 You specialize in obesity, but also hormone optimization, helping people have healthy hormones.
00:01:45.880 So they live a flourishing life.
00:01:47.620 And today I like to talk about hormones, particularly male hormones.
00:01:51.840 I think when most people think about male hormone optimization, they think about testosterone,
00:01:57.420 which is obvious why you do that.
00:01:59.980 We're going to dig deep into testosterone today.
00:02:01.820 But are there other hormones that affect male health that people often overlook?
00:02:07.320 There certainly are.
00:02:08.800 So even testosterone in and of itself, there's nothing unique about it compared to other androgens.
00:02:15.040 There's just one androgen receptor.
00:02:17.160 Testosterone just happens to be the most well-known androgen.
00:02:20.680 So there's DHEA, which is a very weak androgen.
00:02:23.160 It's produced by the adrenal glands, which are small glands above the kidney.
00:02:26.520 There's DHT, which is dihydrotestosterone.
00:02:29.520 This is a very strong androgen.
00:02:31.380 You don't have as much of it as testosterone, but it's vitally important for what's called
00:02:36.040 secondary sexual characteristic development, like the deepening of the voice,
00:02:40.060 growing facial hair, those secondary sexual characteristics, which are vital.
00:02:45.720 And also, I think people often overlook estrogen plays a role in male health.
00:02:50.360 Certainly.
00:02:51.260 Testosterone aromatizes and directly converts to estrogen.
00:02:54.320 So the way to think about estrogen is the more estrogen, the better for your health,
00:02:59.960 because it prevents things like heart attacks at a correct ratio to where you feel good.
00:03:05.940 Okay.
00:03:06.080 So we got to have some estrogen in there at the right balance.
00:03:09.660 And then there's another hormone called SHBG.
00:03:13.620 What does that do?
00:03:14.340 So SHBG is also known as androgen binding globulin.
00:03:18.120 It's a protein.
00:03:19.000 It's made in many places.
00:03:20.460 The liver makes most of it, but the testes also make some of it.
00:03:25.060 And SHBG stands for sex hormone binding globulin.
00:03:28.300 It most strongly binds DHT, and then it binds testosterone relatively strongly.
00:03:33.800 DHEA weaker than that.
00:03:35.320 And then estradiol, which is your main estrogen, even weaker than that.
00:03:39.180 So think of this as regulating all of the hormones and keeping them more stable.
00:03:44.260 The higher the SHBG, the more stable the level will be.
00:03:48.100 Men produce a lot of testosterone during sleep.
00:03:50.780 So the level is generally much higher in the morning.
00:03:53.420 But if you have a very low SHBG, you'll crash.
00:03:56.620 And you can actually have deficient levels of testosterone in the evening routinely.
00:04:00.480 But normal levels in the morning, if you don't have enough SHBG,
00:04:03.940 the most common cause of an SHBG deficiency is insulin resistance,
00:04:07.940 which is often due to too many calories or too many carbohydrates and sugar.
00:04:12.720 So what's interesting about all those hormones is they interact with each other.
00:04:15.540 It's a complex system.
00:04:16.760 So if you raise the level on one, one might go down or up.
00:04:20.200 So I think a lot of guys, they get too focused on,
00:04:22.340 well, I got to increase this one thing or reduce this one thing.
00:04:25.040 Well, if you do that, you're going to have these cascading effects that might not be optimal.
00:04:29.660 Correct.
00:04:30.020 I actually heard an advertisement from a TRT clinic this morning, and it said,
00:04:34.740 new study shows that men with low testosterone are more prone to cardiovascular disease and early
00:04:42.040 death and diseases of aging.
00:04:44.100 And I thought to myself, this is odd because they're implying that you need testosterone
00:04:49.400 replacement to prevent this.
00:04:51.420 But of course, that is a logical fallacy because just replacing the testosterone without figuring
00:04:57.800 out what's actually causing it in the first place.
00:04:59.820 Not that TRT is wrong, but you need to figure out what the cause of it is and then address it.
00:05:05.280 Okay.
00:05:05.740 And I hope we can talk about TRT because I know a lot of guys are thinking about doing it,
00:05:09.640 or maybe they are doing it, and they might have questions about that.
00:05:12.140 Let's talk about testosterone.
00:05:13.680 So there's two ways to measure testosterone, or two measurements of testosterone that I read about.
00:05:18.320 One is total testosterone and free testosterone.
00:05:21.620 So first, what's the difference between the two?
00:05:23.220 And as a clinician, is there a particular number you focus on?
00:05:28.160 Yeah.
00:05:28.580 So total testosterone is a total amount of testosterone, whether it's bound or unbound.
00:05:33.940 When testosterone is bound, it in general does not bind the androgen receptor, which is on the
00:05:39.800 X chromosome.
00:05:40.700 And total testosterone includes a testosterone bound to albumin, which is the main protein in
00:05:45.900 the blood, and also SHBG, which we talked about earlier.
00:05:49.200 But free testosterone, or any free androgen, is what is going to be what is actually binding
00:05:56.080 to the receptor, and then it takes it into the nucleus of the cell, and then it binds
00:06:01.060 to DNA to cause what's called gene transcription.
00:06:04.100 So the androgen receptor gene that's on the X chromosome is then mostly activated by free
00:06:11.100 testosterone.
00:06:12.540 Oddly enough, sometimes I make the analogy of plumbing.
00:06:15.740 So you have a pipe, that's your bloodstream that takes testosterone everywhere, and then
00:06:19.860 you have different types of cells, for example, a muscle cell or a brain cell or a germ cell
00:06:26.340 in the testicle or a somatic cell in the testicle, which we don't have to get into.
00:06:30.960 But anyway, the free testosterone level can be very different in the bloodstream, which
00:06:35.300 is where we measure it on a blood test, versus inside the cell.
00:06:38.920 So it is possible to have symptoms of low testosterone because you don't have enough androgen
00:06:43.440 in the cell but have a normal level in the blood, it's rare, but it's possible, and the
00:06:48.100 opposite is possible, to have a low level in the blood but still have enough inside the
00:06:53.860 cell that's free to be causing normal gene transcription.
00:06:57.880 Okay, so just to recap there, total testosterone is made up of bound and unbound testosterone.
00:07:03.580 Bound testosterone can be bound to albumin or SHBG.
00:07:08.220 And then when it's bound to those things, it can't attach to the androgen receptor in
00:07:12.800 the cell.
00:07:13.640 And so it can't be, can't have those changes on the cell.
00:07:17.280 Free testosterone, unbound testosterone is free testosterone.
00:07:20.340 So as a clinician, when you do a blood test on a patient, like what number is more important
00:07:26.320 to you?
00:07:26.780 Like which one are you going to be focusing on more?
00:07:29.340 Is it the free or the total?
00:07:30.600 I think both are equally important for athletic purposes, for muscle building purposes.
00:07:38.120 Usually that's more correlated with free testosterone level.
00:07:42.080 However, symptoms and how you feel is usually correlated more with total.
00:07:46.300 Insurance companies and academic societies usually put more weight into total testosterone,
00:07:52.120 partly because free testosterone are often measured inaccurately.
00:07:55.260 So often it's more accurate to calculate your free testosterone using your total testosterone
00:08:01.500 and your SHBG.
00:08:03.620 And then you estimate what your free testosterone is.
00:08:06.500 Some societies say low testosterone is often best treated if you have one have symptoms and
00:08:13.480 then two also have a testosterone below about 400.
00:08:16.980 That's what the urologists say.
00:08:18.720 Most other societies go by 300.
00:08:21.160 And I tend to agree with the level of 400 with a caveat if you have significant symptoms
00:08:27.020 and with a second caveat if you cannot improve that naturally in any way after identifying
00:08:33.040 the root cause.
00:08:34.200 Okay.
00:08:34.280 I want to dig more into diagnosing low testosterone because there's lots of commercials out there.
00:08:38.840 You just mentioned one or these businesses popping up where you can just go in and get
00:08:43.060 a blood test and like, hey, you got low T.
00:08:45.200 Here's testosterone.
00:08:46.460 And maybe they don't.
00:08:47.860 So you mentioned two things you look at to diagnose low testosterone.
00:08:51.700 You're going to do blood work.
00:08:53.160 And if it's below 400, coupled with if the patient is reporting symptoms of low testosterone,
00:09:00.260 we'll talk about the symptoms of low testosterone here in a bit.
00:09:02.560 Let's talk about blood work.
00:09:03.540 I think a lot of guys out there, they think it's a panacea.
00:09:05.740 If you just take a test, you take the test and it says, oh, well, your T is at 400.
00:09:10.280 They're like, well, I got low T.
00:09:11.860 Why isn't one blood test alone sufficient to diagnose low testosterone?
00:09:16.300 In general, testosterone levels can have what's called outliers.
00:09:23.360 It's the statistical phenomenon, but it's especially true of testosterone where you
00:09:27.480 could check it one time and your testosterone that morning could be low because the last
00:09:32.320 two nights you've had poor sleep and poor diet and other lifestyle factors.
00:09:37.380 Males that are generally seeking a TRT prescription know this very well because there's various things
00:09:42.180 that you can do to artificially make your testosterone level look low that morning.
00:09:47.880 So in general, the recommendation is to recheck it two to three times after, you know, good
00:09:52.800 night of sleep and normal diet and whatever you're doing normally, not after you've dieted
00:09:57.940 down to 7% body fat to do an ultra marathon or bodybuilding show, then your testosterone is
00:10:03.300 certainly going to be low.
00:10:04.360 But when you're at a healthy body fat and there's not an artificial something else that
00:10:09.200 is going to make your testosterone look low, there's a runner.
00:10:13.200 His name is Nick Bear, and he also is doing a bodybuilding show.
00:10:16.880 And I saw that he got his total testosterone checked and he's a healthy guy.
00:10:20.200 I'm not sure what his baseline testosterone is.
00:10:22.280 And his total testosterone was right at 100 before his bodybuilding show.
00:10:27.500 So that was obviously secondary to the caloric deficit.
00:10:29.820 That wouldn't necessarily count as a testosterone reading that you could put stock in assessing
00:10:35.420 TRT or not.
00:10:36.980 But for most people, they probably won't be in a scenario like that.
00:10:41.100 But it is important to get at least two readings.
00:10:44.180 If you've been sick before, then maybe just postpone the blood test by a week.
00:10:48.080 That way you get an accurate reading.
00:10:50.460 Let's move on to the symptoms.
00:10:51.560 So you do the blood test.
00:10:53.420 What symptoms are you looking for to diagnose low testosterone?
00:10:57.880 Yeah, could be through any system.
00:10:59.320 So it could be anything from depression, anxiety to low libido is certainly classic.
00:11:05.620 Low muscle mass is not really one that we look for.
00:11:08.820 Testosterone levels that are naturally produced are not as correlated as people would think with
00:11:16.200 body composition and muscle mass and athletic performance.
00:11:19.760 So it's not uncommon to see a pretty high level athlete have a total testosterone of, you know,
00:11:24.860 let's say 450 and let's say someone that has very low muscle mass and maybe even 20, 22% body fat to have
00:11:33.740 a total testosterone of a thousand.
00:11:35.840 There's not as much correlation.
00:11:37.020 But other things that you would look for seriously is, for example, erectile dysfunction, sexual health in general, sperm production.
00:11:45.560 So if there's a patient that is having even subfertility, just a little bit of trouble getting pregnant,
00:11:50.320 that individual should certainly have a test of his testosterone as well.
00:11:54.880 So with low libido, how does a guy know if he has low libido?
00:11:59.540 Because that seems like it'd be pretty subjective.
00:12:02.220 Yeah.
00:12:02.480 Libido obviously has a lot of psychosocial factors as well.
00:12:06.680 It's usually taken at a patient's word.
00:12:10.740 And a lot of times when you're testing these patients, you've known the patients for a while.
00:12:14.320 Sometimes you haven't.
00:12:15.180 But if they're telling you that it's low relative to what it usually is and no other factors of change, for example,
00:12:23.880 you know, they've been married to the same person for five years.
00:12:26.860 They're not actively going through problems in the marriage.
00:12:29.780 There's not something else that would be affecting the libido.
00:12:32.540 So that would usually come up in the social history.
00:12:35.020 When you do a history and physical on a patient, it is important to dig into the social history to make sure
00:12:40.480 there's not something else that is affecting the libido.
00:12:43.520 So besides low libido, maybe lack of drive, what are the consequences of suboptimal male hormone levels, like chronically?
00:12:52.260 Is it going to affect your cardiovascular system?
00:12:54.240 Is it going to affect cancers?
00:12:56.320 Does it affect things like that?
00:12:58.080 It will.
00:12:58.940 If someone is significantly hypogonadal for a long time, they're at much higher risk of osteoporosis,
00:13:06.180 which leads to bone fractures and even mortality as well.
00:13:09.640 They're at higher risk of neurodegenerative disease, likely largely due to low estrogen.
00:13:15.280 If you don't have a lot of testosterone, you're probably not converting a lot of it to estrogen.
00:13:19.300 And if you're not doing that, then you're also at risk of cardiovascular disease.
00:13:23.500 Estrogen is very cardioprotective and helps with the production of good cholesterol to help take cholesterol out of the plaque.
00:13:30.020 So they've done studies and you look at one group of people that have true hypogonadism, which is generally two levels under 264 or so.
00:13:39.640 And then one group you give TRT and then one group you don't give TRT.
00:13:45.540 You would think that the group that you give TRT would have a shorter lifespan because androgens do cause excess production of quote unquote bad cholesterol.
00:13:55.500 They do increase a particle called ApoB, which is the most important one to watch for cardiovascular risk.
00:14:01.120 But the group that you give TRT actually has less heart attacks and strokes.
00:14:04.600 Right, because what you were saying before, the testosterone creates estrogen and then the estrogen protects the heart.
00:14:11.000 So let's talk about the causes of low testosterone.
00:14:13.660 What can be behind low T?
00:14:15.960 Most commonly metabolic syndrome.
00:14:18.620 So excess calories, excess carbs, insulin resistance, high fasting insulin leads to the liver not producing SHBG.
00:14:27.320 So you might be producing a decent amount of testosterone, but it's being metabolized so fast that it's difficult to use.
00:14:33.980 That's most common.
00:14:35.140 The second most common, I think, is sleep apnea or obstructive sleep apnea.
00:14:39.760 Obviously, that kind of goes hand in hand with metabolic syndrome, but often it goes hand in hand with PTSD.
00:14:45.540 I saw a study on young men that had just gotten out of the military and they had been diagnosed with PTSD and they tested them all for sleep apnea.
00:14:56.080 And something like 80% of them had sleep apnea and they were all under a BMI of 25.
00:15:02.360 So there's certainly a lot of stress component as well.
00:15:05.980 The limbic system includes places like the hypothalamus and the amygdala and downstream to that is the hippocampus and the amygdala.
00:15:13.320 Downstream of those is the hypothalamus.
00:15:16.120 And that's some of the places of the brain that are involved in sleep regulation and breathing.
00:15:22.060 So the theory is that apneic episodes don't just come from having a huge neck and excess body fat, but there are other factors like trauma at play.
00:15:32.040 And when you have a patient with severe sleep apnea, they have a score called an AHI score.
00:15:40.100 And if that score is very high, like 100 or 200, you almost always see deficient testosterone levels.
00:15:47.040 Okay, so having metabolic syndrome, being overweight, sleep apnea, any other causes of low testosterone?
00:15:52.920 Yeah, so theoretically, xenoestrogens could be a cause of low testosterone.
00:15:58.700 These are things like phthalates.
00:16:00.240 These are also things like bisphenol A, also known as BPA.
00:16:03.520 You might see BPA-free on water bottles from time to time.
00:16:07.180 These do bind various estrogen receptors and are likely suppressive.
00:16:12.260 By suppressive, I just mean they shut down the production of the hormones that lead to testosterone production to some degree.
00:16:19.160 Heat damage is also kind of an honorable mention.
00:16:22.840 Some people might be familiar with what varicose veins are.
00:16:25.760 Varicocele is where there's varicose veins in the scrotum.
00:16:28.200 And some people with varicocele can have venous cooling very well.
00:16:32.880 The testes want to be about 91 to 92 degrees, where the body is 98.6 degrees.
00:16:38.060 So if you can't keep your testes at 91 or 92, then you're going to have less testosterone production and less sperm production.
00:16:46.440 And in the more severe cases, you'll have atrophy, which is shrinking because, think about them as factories.
00:16:52.880 If you're not using the factory, they start to shut down.
00:16:56.600 And besides these lifestyle factors, environmental factors, you could also have just an issue with your pituitary system, right?
00:17:02.760 You might have a tumor or something in your pituitary gland that's dysregulating the release of hormones.
00:17:07.900 Correct.
00:17:09.360 I suppose that would be likely one of the more common, less modifiable risk factors.
00:17:15.040 There's not a lot that you can do about that.
00:17:16.640 You can take supplements like vitamin B6 or like vitamin E.
00:17:20.560 But a lot of times, pituitary microadenomas or even macroadenomas, basically, it's a small tumor in the brainstem.
00:17:29.180 The pituitary gland is where you make a lot of different hormones, like growth hormone and like LH and FSH.
00:17:34.900 But LH is the main hormone that's produced there that leads to testosterone release.
00:17:39.840 So there's two different types of hypogonadism.
00:17:43.400 There's primary and secondary.
00:17:45.080 So primary is where the testes are not functioning.
00:17:49.580 And then secondary, think about it, it's two steps instead of one step.
00:17:53.340 So the LH can be low in secondary hypogonadism.
00:17:57.040 And if your LH is very low and a hormone like prolactin or IGF-1 is very high,
00:18:03.020 then that might be a sign of a pituitary microadenoma, in which case you need an MRI.
00:18:08.980 And LH, that's luteinizing hormone, correct?
00:18:11.380 Correct.
00:18:11.680 LH is luteinizing hormone.
00:18:13.260 FSH is follicle-stimulated hormone.
00:18:15.780 They do cross-talk to some degree.
00:18:18.100 But LH mostly helps with testosterone production and mostly binds to the Leydig cell in the testicle.
00:18:24.700 And FSH mostly binds in the seminiferous tubules and helps with spermatogenesis.
00:18:31.200 So, I mean, listeners have probably heard reports that T levels in men have been declining in the past few decades.
00:18:36.320 Do we know what's causing this sort of general decline?
00:18:39.140 Is it just all these lifestyle, like people are getting fatter, they're not sleeping, they're stressed, and the stuff in the environment,
00:18:44.420 is that kind of what we've decided is the cause of the lower T levels?
00:18:48.800 The various causes that we've already discussed are likely the primary causes of what is causing declining testosterone levels.
00:18:55.560 But I think there is another factor, and a lot of that has to do with what I'd call epigenetic drift.
00:19:04.240 Some people might call it natural selection.
00:19:06.520 I might call it unnatural selection, where individuals with higher testosterone levels are no longer being selected for as early.
00:19:18.160 And also, a lot of individuals are having kids later on in life.
00:19:23.280 For example, in their 30s or even 40s, when you might have very different maternal and paternal hormone profiles.
00:19:30.760 That's one of the reasons why I recommend if men are taking medications like finasteride or dutasteride,
00:19:37.000 that they stop their finasteride 90 days before attempting conception.
00:19:41.260 And they stop dutasteride, depending on what dose they are, usually six months before conception.
00:19:45.740 By the way, spermatogenesis takes about 60 days or two months.
00:19:50.000 That way, they have enough time to wash out before they start producing the sperm in the germ cells,
00:19:55.520 so that they wouldn't pass down any epigenetic changes to potential offspring.
00:20:01.300 Okay, so maybe the idea is that, again, this is theoretical, right?
00:20:04.820 The testosterone increases aggression and risk-taking behaviors,
00:20:08.560 and that's not as adaptive in our safe, high-tech, modern landscape.
00:20:13.200 So men with lower testosterone might be more successful these days,
00:20:17.820 and women choose those men for their partners.
00:20:20.480 And then when they have children, the men pass down his genes,
00:20:24.560 and then his children have lower testosterone too.
00:20:27.840 And that just perpetuates just lower testosterone in the male population overall.
00:20:33.000 Also, this idea of selection.
00:20:36.140 I've heard that, I read this somewhere, correct me if I'm wrong on this,
00:20:40.520 that women on birth control, they're not attracted to higher testosterone men.
00:20:45.640 Is that true?
00:20:46.720 Yeah, that is one of the major players of what I would call unnatural selection.
00:20:52.740 Another interesting unnatural selection, I suppose,
00:20:56.700 if you look at, not very recently, but the World War,
00:20:59.500 certainly in World War I and World War II,
00:21:01.400 or in the Korean War or Vietnam War,
00:21:04.600 but especially wars that, even if a war has a draft,
00:21:09.420 the individual that has higher testosterone and also more sensitive androgen receptors,
00:21:15.580 so this is probably true throughout all of human history,
00:21:19.020 you would, and this obviously cannot be proven scientifically,
00:21:22.240 but theoretically, that individual would be more likely to volunteer
00:21:25.700 to go to the front line or to very risky positions.
00:21:30.000 And if that male passes away at age 18 or age 19,
00:21:34.740 then that is likely a fecundity rate of zero,
00:21:38.780 so no offspring from that individual,
00:21:40.580 and then you start to have genetic drift.
00:21:42.940 Okay, so again, this is theoretical.
00:21:44.580 What you're saying is that men with very high testosterone,
00:21:47.660 they're going to take more risk,
00:21:49.120 and in doing so, that may take them out of the gene pool
00:21:51.960 by taking those risks.
00:21:53.340 And there's more opportunity for that sort of risk-taking
00:21:55.940 during big global conflicts like the World Wars, right?
00:21:59.100 More high-T men die,
00:22:00.600 they lose the chance to reproduce and pass on their genes,
00:22:03.620 and then that just contributes to the declining testosterone in men in general,
00:22:07.900 and that's going to have echoes through the generations.
00:22:11.700 And on top of that,
00:22:13.180 we just have selection factors going on in the mating market as well.
00:22:18.300 Yes, and it's not like it's an be-all, end-all,
00:22:22.460 you know, all-or-nothing.
00:22:24.040 You select for high testosterone or you select for low testosterone.
00:22:27.020 There's a lot more psychosocial factors at play,
00:22:29.680 but we are certainly seeing that there's likely a decline in testosterone
00:22:35.240 even a bit more than could be accounted for
00:22:37.680 by just metabolic syndrome and sleep apnea.
00:22:41.460 Maybe things like heat damage to the testicle,
00:22:44.440 maybe things like xenoestrogens are playing some part in this,
00:22:49.620 but we'll probably never know,
00:22:51.800 but it's very fun to speculate about it.
00:22:53.600 Well, the heat damage to the testicle,
00:22:54.940 like what would cause,
00:22:56.060 is it like keeping your laptop on your lap sitting down a lot?
00:22:58.360 Is that, would that cause heat damage?
00:23:01.200 Probably not significantly enough,
00:23:03.140 but if you already had a varicocele
00:23:05.180 and you already spent an hour in the jacuzzi,
00:23:08.820 keeping your really hot laptop and phone
00:23:11.280 directly over your scrotum is certainly not going to help.
00:23:14.060 I suppose someone could prove this at some point.
00:23:16.000 They've actually done a lot of studies
00:23:17.880 where they look at the scrotal temperature
00:23:19.880 and they've randomized two groups of usually college students.
00:23:24.800 And one group they have where basically like a sock
00:23:28.720 around their scrotum that has something really warm in it.
00:23:32.100 So they warm up the scrotum artificially to 98 degrees
00:23:36.980 instead of 91 or 92 degrees.
00:23:39.780 And in the individuals that don't have varicocele,
00:23:42.720 they can still overcome that heat damage
00:23:45.520 because their venous pooling mechanism
00:23:48.080 is so good at buffering that heat damage.
00:23:50.940 So that did not affect their testosterone production
00:23:53.460 and it did not affect their spermatogenesis.
00:23:56.700 However, in individuals that already have impaired venous cooling,
00:24:00.340 for example, with varicocele or varicose veins,
00:24:03.140 then it did.
00:24:04.440 We're going to take a quick break
00:24:05.480 for a word from our sponsors.
00:24:06.580 And now back to the show.
00:24:12.920 Let's talk about optimal levels of testosterone.
00:24:15.320 So below 400,
00:24:16.860 and if you're experiencing low testosterone symptoms,
00:24:19.120 that's not good.
00:24:20.820 Is there an optimal level?
00:24:21.980 Is there a level that guys should reach for?
00:24:24.320 Or is it going to differ from man to man?
00:24:26.640 It certainly differs,
00:24:27.520 but that's kind of an easy answer.
00:24:29.240 So I'll get into it more than that.
00:24:31.120 A lot of times people have told me
00:24:33.120 that I say individualized.
00:24:34.580 I say that word a lot because health is individualized.
00:24:38.220 We are all unique.
00:24:39.640 We have different genetics.
00:24:41.140 We have different epigenetics
00:24:42.240 and we have different growth and development past that as well.
00:24:46.100 But for most men,
00:24:47.780 an optimal testosterone level is between about 500
00:24:50.360 and as high as you can go naturally.
00:24:53.420 So, you know, there is some individuals
00:24:54.860 with a total testosterone of 1500.
00:24:57.480 They almost always have really high SHBG.
00:25:00.920 So a lot of times their free testosterone is only 20 or 25.
00:25:04.640 Between about 550 and whatever you can produce
00:25:07.340 top-in endogenously naturally without medication.
00:25:11.000 But you all say it could be lower.
00:25:12.300 I mean, you mentioned there are athletes who are at 450
00:25:14.620 and they're healthy.
00:25:16.400 So if you get a blood test and it's below 500 a little bit,
00:25:19.460 you probably, I mean,
00:25:20.360 I guess you shouldn't worry too much about it
00:25:22.000 if you're not experiencing any symptoms.
00:25:24.520 Correct.
00:25:25.260 Okay.
00:25:25.880 That's good to know.
00:25:26.960 So let's say a patient comes to you
00:25:28.520 reporting symptoms of low T.
00:25:29.920 You do a series of blood tests that show,
00:25:32.260 yeah, that your T levels are low.
00:25:33.860 They're below 400.
00:25:35.320 What's your first line of attack
00:25:36.940 in helping this patient get his T levels up?
00:25:40.440 First thing to look at would be LH and FSH.
00:25:43.020 If those are really low,
00:25:44.040 then I'm worried about the pituitary or the brain.
00:25:46.280 If those are really high,
00:25:48.000 then I'm worried about the health of the testicles.
00:25:51.220 If they're in between,
00:25:52.840 then I look for another pathology,
00:25:54.900 like diabetes, metabolic syndrome,
00:25:56.880 insulin resistance, sleep apnea, et cetera.
00:25:59.600 I also look at prolactin and IGF-1.
00:26:02.240 Make sure you assess their tumor risk.
00:26:04.340 And then I also look at estradiol.
00:26:05.960 If it's a very high estradiol,
00:26:07.760 then estradiol is likely what is suppressing
00:26:10.000 the production of LH from the pituitary.
00:26:13.180 So you have estradiol, which is your main estrogen,
00:26:15.780 which is causing less testosterone production.
00:26:18.480 And in that case,
00:26:19.300 I look at things like alcohol consumption
00:26:21.000 that can upregulate aromatase
00:26:22.560 or consumption of excess calories or fat
00:26:25.060 that can upregulate aromatase,
00:26:27.260 which converts testosterone to estrogen, by the way.
00:26:30.340 So those are the first things.
00:26:32.460 Beyond that, what are you looking at?
00:26:34.700 Beyond that, I'd like to, if pertinent, do an exam.
00:26:38.420 Make sure, especially if this individual is,
00:26:41.360 you know, developing,
00:26:42.640 if they're an adolescent or whatnot,
00:26:44.440 you need to make sure that they're
00:26:45.380 through all the Tanner stages,
00:26:46.940 basically Tanner stages one to five,
00:26:49.300 five is done.
00:26:50.020 When you're essentially adult growth and development
00:26:52.980 to make sure that they don't have some
00:26:55.740 unusual or unlikely syndrome.
00:26:58.940 And then after that,
00:27:00.700 I'd like to look at their fasting insulin,
00:27:03.580 their A1C,
00:27:05.140 see if there's something that I can correct.
00:27:06.480 I look at their cortisol.
00:27:07.480 If their cortisol is high,
00:27:08.680 then there's a lot of lifestyle factors
00:27:10.760 and also supplements
00:27:11.720 that can help control cortisol
00:27:14.020 like ashwagandha or emodin.
00:27:15.800 I look at their prolactin.
00:27:17.500 So if their prolactin is just a little bit high,
00:27:19.360 then maybe I do start them on some vitamin B6
00:27:21.680 or some vitamin E.
00:27:23.760 If their estrogen is high,
00:27:25.320 maybe I start them on some calcium D-glucarate
00:27:27.180 that helps with estrogen,
00:27:28.740 glucuronidation and metabolism.
00:27:30.300 It basically helps you excrete it through your stool.
00:27:33.440 And then repeat labs in one, two,
00:27:36.700 maybe even three months
00:27:37.660 and see if we can improve those things
00:27:39.960 along with, as always, diet and exercise.
00:27:43.300 Okay.
00:27:43.440 So it sounds like the first line of attack,
00:27:45.220 if it's not a pituitary problem,
00:27:47.260 you're going to be primarily doing lifestyle changes, right?
00:27:50.200 Quitting drinking, getting better sleep,
00:27:52.820 diet, exercise to help get
00:27:54.520 that insulin sensitivity back online.
00:27:57.480 So yeah, lifestyle stuff would be
00:27:58.600 the first line of attack.
00:27:59.560 And then will it take maybe one to two months
00:28:02.300 before you start seeing results from that?
00:28:04.460 Yeah, often it does.
00:28:05.480 A lot of times you feel better the first week
00:28:07.580 and a lot of times your testosterone production
00:28:09.560 recovers very quickly.
00:28:11.880 But occasionally I use medications as well.
00:28:14.420 So some people utilize a short course of HCG,
00:28:18.020 which essentially binds the LH receptor,
00:28:20.220 takes the place of LH.
00:28:21.740 And occasionally I'll utilize very short courses.
00:28:24.600 By very short, I mean a week
00:28:25.960 or maybe two weeks of selective estrogen receptor modifiers,
00:28:29.640 or sometimes longer in the right patient,
00:28:32.700 especially very young patients
00:28:34.200 that you're trying to stimulate endogenous production.
00:28:37.420 These are often patients that desire fertility
00:28:39.700 within the near to mid near future.
00:28:42.920 Besides diet, exercise, sleep, managing stress,
00:28:47.020 you mentioned a few supplements
00:28:48.160 that you recommend men taking
00:28:49.380 to optimize male hormones.
00:28:50.820 Are there ones that you recommend for just any guy
00:28:53.280 who maybe they don't have any problems with their testosterone,
00:28:55.980 but they just want to feel good?
00:28:57.580 Are there ones that you like and that are safe?
00:29:00.520 Creatine, five to 10 grams a day would be a great start.
00:29:03.480 L-carnitine would be a consideration,
00:29:06.360 especially if they're interested
00:29:07.680 in athletic performance optimization
00:29:09.660 or body composition optimization.
00:29:12.760 L-carnitine would be reasonable.
00:29:14.040 Consider checking a TMAO
00:29:15.480 to make sure that it doesn't convert to that
00:29:17.500 and too high of a rate.
00:29:19.360 Another reasonable addition,
00:29:20.740 if someone has high estradiol,
00:29:23.160 would be calcium deglucarate
00:29:25.060 to make sure that they're binding up extra estrogen
00:29:27.320 and excreting it.
00:29:28.940 I've heard that boron can impact testosterone.
00:29:31.560 How does boron increase T levels?
00:29:33.900 Boron works okay for people
00:29:36.120 with really high SHBGs.
00:29:38.000 It increases free testosterone
00:29:40.480 by decreasing SHBG.
00:29:42.580 The effect wears off to some degree
00:29:44.420 if you take boron for a very long period of time.
00:29:46.840 If you have very low levels
00:29:48.940 or you're insufficient or deficient in boron,
00:29:51.040 it works extremely well.
00:29:52.640 And a lot of people consume dates or raisins
00:29:55.480 because they tend to be relatively high in boron.
00:29:59.020 There's another something I've been hearing about lately,
00:30:00.500 Tunggat Ali, I think that's how you pronounce it.
00:30:02.180 What's going on with that one?
00:30:03.300 Tunggat Ali is also known as Long Jack.
00:30:06.740 So Tunggat's active ingredients are Uri peptides,
00:30:10.560 one of which is Uri comanone.
00:30:12.880 And Tunggat is helpful
00:30:14.760 because it upregulates a couple different enzymes
00:30:17.060 in the stroitogenesis pathway.
00:30:19.240 There's been plenty of human study on it
00:30:21.400 with mixed results.
00:30:23.060 And it looks like the cause of the mixed results
00:30:25.580 is sometimes people have great activity of those enzymes.
00:30:29.000 So that's not the rate limiting step
00:30:30.800 in testosterone production.
00:30:31.940 So think of it as a signal.
00:30:34.480 Think of your testicles as a factory.
00:30:36.720 Tunggat is a signal to that factory
00:30:38.320 to ramp up production.
00:30:40.400 But if your factory is already operating
00:30:42.260 at maximum capacity
00:30:43.460 or it's limited by something else,
00:30:46.020 then that's not going to improve
00:30:48.160 your testosterone level.
00:30:49.720 Tunggat works on very similar enzymes
00:30:51.660 that are also upregulated by insulin and IGF-1.
00:30:55.860 So in general, if you're in a caloric deficit
00:30:57.920 or if you're trying to lose weight or body fat,
00:31:00.320 Tunggat will work better.
00:31:01.740 If you have a low fasting insulin
00:31:03.340 or a lower end IGF-1,
00:31:06.280 Tunggat will also likely work better.
00:31:08.140 And I've seen this anecdotally as well.
00:31:10.160 A couple of years ago,
00:31:10.700 I remember ZMA was a big supplement
00:31:12.660 that was pushed for increasing testosterone levels.
00:31:15.400 Anything to that?
00:31:17.200 ZMA is very reasonable to add
00:31:18.860 if you have a low ALKFOS.
00:31:20.520 So if you look at your CMP,
00:31:22.560 which is your metabolic panel,
00:31:24.300 there'll be an enzyme called alkaline phosphatase.
00:31:26.740 It's alkaline phosphatase along with GGT
00:31:29.460 or two intracellular enzymes.
00:31:31.720 And the lower these two are,
00:31:33.080 the more likely you are to have insufficient levels
00:31:35.560 of zinc and magnesium.
00:31:37.280 That's why when I have input to various companies
00:31:39.820 designing a supplement to optimize testosterone,
00:31:43.860 I almost always put in zinc,
00:31:45.600 magnesium, and vitamin D.
00:31:47.500 You just want to make sure
00:31:48.260 these aren't the right limiting stuff.
00:31:49.860 Think about trying to optimize your testosterone
00:31:51.660 as like trying to get into a fraternity.
00:31:53.360 You're not just making best friends
00:31:56.500 with one of the people
00:31:57.980 and then just hoping
00:31:58.840 that nobody else will blackball you.
00:32:00.860 You want to make sure
00:32:01.420 that you address each individual
00:32:03.020 because if you,
00:32:04.140 like let's say you forget your vitamin D
00:32:05.680 and forget your zinc,
00:32:06.640 you're deficient in zinc,
00:32:07.920 you're deficient in vitamin D.
00:32:09.580 Those two things will hold you back.
00:32:12.060 Once you start down this path
00:32:13.200 of increasing your testosterone
00:32:14.460 or getting them optimized,
00:32:17.060 is there any benefit to getting them higher?
00:32:19.260 So like let's say you started off at 400,
00:32:21.980 you had low T symptoms.
00:32:22.880 And then through lifestyle changes
00:32:24.840 and maybe taking some supplements,
00:32:26.160 you bump it up to like 700.
00:32:29.880 Are you going to get any more benefit
00:32:31.360 from testosterone
00:32:32.340 by getting it up to 800 or 900?
00:32:35.100 Past about 600,
00:32:36.500 there's little to no benefit
00:32:37.540 other than bragging rights.
00:32:39.140 At what point would you have a patient
00:32:41.120 go on testosterone replacement therapy?
00:32:44.140 At any point when the risks outweigh the benefits
00:32:46.800 and they understand both the risks
00:32:49.580 and the benefits in their own terms.
00:32:52.260 So what are the risks of TRT?
00:32:54.500 Yeah.
00:32:54.840 One of the risks is
00:32:56.100 it causes more fluid retention and swelling.
00:32:58.320 One of the risks is
00:32:59.260 if you hyperconvert to estrogen,
00:33:01.140 estrogen will then bind to the liver
00:33:02.660 and cause more SHBG and platelet production.
00:33:05.740 And if your platelets go very high
00:33:07.180 past a certain point,
00:33:08.160 we know that people on oral estrogen,
00:33:11.040 the blood clot risk is associated
00:33:12.440 with how high their platelets and SHBG go.
00:33:15.480 It's likely the same for TRT.
00:33:17.180 So if you go on TRT
00:33:19.560 and you go into a huge bulk
00:33:20.800 and you start consuming a bunch of alcohol
00:33:22.420 and your platelets skyrocket,
00:33:24.260 then it is going to increase
00:33:25.940 your blood clot risk.
00:33:27.880 So TRT is not in and of itself
00:33:30.000 going to improve health.
00:33:31.400 It's just going to be a tool
00:33:32.620 to help you achieve a lot of your goals.
00:33:36.120 Another risk of testosterone is
00:33:38.640 if people have heard of medications
00:33:40.280 called statins,
00:33:41.180 those work by decreasing the activity
00:33:43.860 of an enzyme called HMG C-way reductase,
00:33:46.940 any androgen including testosterone
00:33:48.880 increases the activity of this enzyme.
00:33:51.820 So often people's cholesterol,
00:33:53.860 and it's not actually cholesterol,
00:33:55.280 it's their lipoproteins,
00:33:56.620 but people's quote unquote
00:33:57.940 bad cholesterol gets worse.
00:33:59.520 That's why we watch that APOB number
00:34:01.240 very closely
00:34:02.080 because we know that APOB
00:34:03.800 is the particle that is going to
00:34:06.080 lead to plaque formation
00:34:07.740 in areas like the coronary artery.
00:34:10.120 And I guess the benefits of TRT
00:34:13.020 is that you'll mitigate those
00:34:14.600 symptoms of low testosterone.
00:34:17.180 Correct.
00:34:18.080 And there's of course other benefits as well,
00:34:19.880 like the benefits of estrogen
00:34:21.020 that we discussed earlier
00:34:22.700 being its cardioprotective benefit.
00:34:25.180 And one of the main benefits of testosterone
00:34:27.940 in a lot of individuals that I see start
00:34:29.700 is they might have it,
00:34:30.960 let's say they have an A1C of 5.7 or 5.8,
00:34:34.060 which is technically pre-diabetes.
00:34:36.200 You're very unlikely to get diabetes
00:34:39.320 on testosterone compared to
00:34:41.300 if you are not on TRT.
00:34:43.540 So a lot of individuals,
00:34:45.200 perhaps they're,
00:34:46.240 I wouldn't say doomed,
00:34:48.020 but very likely to get diabetes.
00:34:50.180 And TRT can make a huge difference,
00:34:52.300 especially when combined
00:34:53.540 with other insulin-sensitizing medications
00:34:55.940 to prevent that.
00:34:57.580 Do you keep people on TRT indefinitely?
00:34:59.420 Is it like once you start,
00:35:00.140 do you keep doing it?
00:35:00.800 Or are there periods where you're like,
00:35:02.160 well, we're going to take you off
00:35:03.140 and see what happens?
00:35:03.920 Or how does that work?
00:35:05.480 Most individuals are on indefinitely,
00:35:08.460 but not everyone.
00:35:09.900 Occasionally, there will be a patient
00:35:11.140 that is profoundly hypogonadal.
00:35:14.000 And the benefit of testosterone
00:35:15.900 at that time is just huge.
00:35:17.680 Let's say it's a patient
00:35:18.540 who has a BMI of 40
00:35:20.560 and they weigh 400 pounds.
00:35:22.980 And they also don't have
00:35:24.300 a huge amount of lean body mass
00:35:25.860 to lose in proportion.
00:35:27.400 Everybody who weighs 400 pounds
00:35:28.620 is going to have a lot of lean body mass,
00:35:30.180 but just less relative
00:35:31.220 to your average person.
00:35:32.160 And they want to maintain
00:35:33.900 as much of that as possible.
00:35:35.060 They need that tool
00:35:36.940 in order to exercise.
00:35:38.520 Even if it's somewhat of a placebo tool,
00:35:40.720 that still helps.
00:35:42.200 So if it gets them
00:35:43.040 having a very healthy lifestyle,
00:35:45.300 they go on that medication.
00:35:46.560 Perhaps they go on another medication
00:35:47.800 like a GLP-1
00:35:48.960 for a short period of time.
00:35:50.820 And then they don't really know
00:35:52.300 what their baseline testosterone is.
00:35:53.780 So maybe after two years,
00:35:55.000 they learn those lifestyle interventions.
00:35:57.500 They very slowly are ready
00:35:59.980 to come off of every medication.
00:36:02.160 And then you can use a medication
00:36:04.500 like HCG
00:36:05.540 to help restore natural production.
00:36:07.740 Perhaps one week of a medication
00:36:09.600 like Inclomiphene or Novodex
00:36:11.380 or even Riloxifene.
00:36:13.020 And then you see
00:36:14.140 what their natural production capability is.
00:36:16.600 You give them a few weeks
00:36:17.660 and perhaps they restore
00:36:19.780 to a total testosterone of 600s,
00:36:21.980 which is likely quite good
00:36:24.560 in that situation.
00:36:25.440 Or perhaps they go down
00:36:26.740 to 100s again.
00:36:28.240 But a lot of people
00:36:29.860 would want that chance
00:36:32.880 to go back to producing
00:36:34.600 their testosterone naturally.
00:36:36.500 And in some cases,
00:36:37.240 it does work.
00:36:38.360 I would say 90% of people
00:36:40.300 that start on testosterone
00:36:41.420 are going to remain on it indefinitely.
00:36:45.020 But I would also say that
00:36:46.560 90% of people
00:36:48.160 that go on testosterone
00:36:49.060 can very likely regain
00:36:51.440 at least their
00:36:53.340 previous level of testosterone
00:36:55.220 if they were to want to come off.
00:36:57.000 Well, here's a question.
00:36:58.600 With female hormone therapy,
00:37:00.540 you might start taking it
00:37:02.440 during menopause
00:37:03.800 to help with symptoms.
00:37:05.680 But at a certain point,
00:37:07.060 you know,
00:37:07.300 like once menopause is over,
00:37:08.960 I think you're supposed
00:37:09.840 to get off those hormones.
00:37:11.640 Does something like that happen
00:37:12.820 for men?
00:37:13.540 Like, I mean,
00:37:13.900 you might do TRT
00:37:15.020 throughout your 50s and 60s.
00:37:16.600 And then at a certain point,
00:37:17.620 you're in your 70s
00:37:18.880 and you're like,
00:37:19.340 well,
00:37:19.680 I don't need to do this anymore.
00:37:21.640 Or are there 80-year-old
00:37:23.020 or 90-year-old guys
00:37:24.120 taking TRT?
00:37:24.920 There are 80-year-old
00:37:26.460 or 90-year-old guys
00:37:27.200 taking TRT.
00:37:28.980 Occasionally,
00:37:29.500 you will do a dose adjustment.
00:37:31.540 It just kind of depends
00:37:32.380 on the situation.
00:37:33.640 But a lot of times
00:37:34.660 when males reach that age,
00:37:37.900 they are less likely
00:37:39.620 to have as much benefit
00:37:41.320 and they are more likely
00:37:42.680 to have slightly more harm.
00:37:44.720 So it's a moving target
00:37:46.440 over time
00:37:47.240 where you get out the scale
00:37:48.640 and you're weighing
00:37:49.260 the risks and the benefits.
00:37:50.940 And at that point,
00:37:51.820 when a patient's already on TRT,
00:37:53.240 you also weigh the risks
00:37:54.500 of how difficult
00:37:56.180 it would be to come off,
00:37:57.280 which is not extremely difficult.
00:37:59.040 But it is difficult
00:37:59.780 because there's medication regimens
00:38:01.320 that you have to go with.
00:38:02.720 And even with those medications,
00:38:04.480 often there is a short period of time
00:38:06.100 when you don't feel great.
00:38:08.160 So we've been talking about
00:38:08.660 optimizing male hormones
00:38:09.940 in grown men.
00:38:10.840 But let's say we got some dads
00:38:12.060 and moms out there listening
00:38:13.140 and they've got boys
00:38:14.040 who are about to start
00:38:15.840 or are in the middle of puberty.
00:38:17.680 What can they do for their sons?
00:38:19.760 What can young guys do
00:38:21.480 to make sure they set themselves up
00:38:23.040 for a lifetime
00:38:24.340 of male hormone optimization?
00:38:27.960 First and foremost,
00:38:29.380 no huge dirty bulk
00:38:30.920 in early adolescence.
00:38:32.300 What I mean by that is,
00:38:33.620 you know, like let's say
00:38:34.340 there's somebody
00:38:34.820 that's trying to put on weight
00:38:36.040 for football
00:38:36.720 or whatever other reason.
00:38:38.600 Can't think of many reasons
00:38:39.420 where it would be worth it.
00:38:40.640 But they're putting on weight
00:38:41.840 and also putting on fat.
00:38:43.880 Adipose tissue in fat,
00:38:46.420 adipose tissue is fat,
00:38:47.400 that is going to increase
00:38:48.760 the conversion to estrogen
00:38:49.860 and estrogen is going to close
00:38:51.600 the growth plates of the bone.
00:38:53.160 So that's going to prevent you
00:38:54.360 from reaching full stature,
00:38:55.660 both in height
00:38:56.960 and other areas
00:38:58.040 of your skeletal developments as well.
00:39:00.260 So that's a great initial recommendation.
00:39:03.480 Thinking about gut health
00:39:04.700 and fiber consumption
00:39:05.660 is also very important.
00:39:07.520 That's going to prevent,
00:39:08.760 again, from over,
00:39:10.120 it's called intrahepatic circulation
00:39:11.660 of estrogen.
00:39:12.920 Estrogen is not necessarily the enemy.
00:39:14.660 In fact, a little bit of estrogen
00:39:16.000 is needed to what's called
00:39:17.160 priming the pituitary
00:39:18.600 in order to fully kickstart adolescence.
00:39:22.320 And that's one of the reasons
00:39:23.060 why boys with very high body masses
00:39:25.880 have higher estrogens.
00:39:27.120 The pituitary gets primed too early
00:39:28.820 and something called
00:39:29.700 precocious puberty is happening,
00:39:31.340 which is too early of puberty.
00:39:33.700 So that's another thing to consider.
00:39:35.560 In addition to that,
00:39:37.280 you want to have a reasonable balance
00:39:39.680 between cardiovascular exercise
00:39:41.260 and resistance training.
00:39:42.580 You certainly want to do both
00:39:44.200 because adolescence can be thought of
00:39:47.040 as your free endogenous steroids.
00:39:51.060 I'll say cycle
00:39:51.840 just because people understand it,
00:39:53.300 but your free endogenous steroid boost
00:39:55.440 where you know you are going to be
00:39:57.000 one, super sensitive
00:39:58.840 to all the androgens that are released
00:40:01.120 that probably most people
00:40:02.540 remember puberty.
00:40:03.520 And you will also be
00:40:04.980 having a lot of androgen around,
00:40:08.320 regardless of what you do,
00:40:09.660 even if your health hasn't been great.
00:40:11.040 So when that endogenous steroid burst happens,
00:40:15.380 that is the perfect time
00:40:17.140 to take advantage of those lifestyle tools
00:40:19.080 to build up very high bone mineral density
00:40:22.260 and very high lean body mass
00:40:24.140 without putting on excess body fat.
00:40:27.240 I imagine young people getting plenty of sleep
00:40:29.320 is important too.
00:40:31.100 Yes, extremely important.
00:40:32.980 And that might be one of the most common causes
00:40:35.540 of suboptimal hormone profiles
00:40:38.620 in adolescence.
00:40:41.100 What about supplementation?
00:40:42.100 Is supplementation something you encourage
00:40:43.700 in young people
00:40:45.140 to optimize their hormones
00:40:46.080 or do you just focus on the diet and exercise?
00:40:49.540 With the oversight of a doctor,
00:40:51.500 I do encourage supplementation
00:40:53.160 if it makes sense.
00:40:54.480 For example,
00:40:55.040 let's say there's a young person
00:40:56.440 and they get a stool test
00:40:58.140 and the beta-glucuronidase enzyme
00:40:59.620 is very high.
00:41:00.660 We know that that individual
00:41:01.660 is just recycling their estrogen
00:41:03.340 over and over again.
00:41:04.540 That makes something like a calcium deglucrate.
00:41:06.620 Or with the oversight of the doctor,
00:41:09.020 maybe even a very low dose
00:41:10.280 of an aromatase inhibitor,
00:41:12.040 a very reasonable addition.
00:41:13.660 And then if you get blood tests,
00:41:14.800 you can actually check the hormones
00:41:15.980 to make sure that they're increasing
00:41:17.600 at the correct rates,
00:41:19.380 that your DHT is optimal,
00:41:21.200 your testosterone is optimal,
00:41:22.440 your estradiol is optimal,
00:41:23.600 your IGF-1 is optimal.
00:41:25.460 And then you can tweak a supplement.
00:41:27.020 Supplements are just like medications.
00:41:29.040 They have pharmacologic effects,
00:41:31.000 so they have an effect on the body
00:41:32.020 and the body metabolizes them.
00:41:34.220 So things like creatine
00:41:35.260 can be very reasonable.
00:41:36.440 Creatine does not affect
00:41:37.540 the development of the kidneys.
00:41:39.140 I did a podcast
00:41:39.800 with my good friend,
00:41:41.660 James O'Hara, recently.
00:41:43.080 We get a lot of questions
00:41:43.860 from pediatricians
00:41:44.860 because the AAP,
00:41:46.380 which is a society of pediatricians,
00:41:48.880 still recommends
00:41:49.740 no creatine supplementation whatsoever
00:41:52.380 up to the age of 18.
00:41:54.300 So not even a 17-year-old.
00:41:56.680 So I just kind of thought
00:41:57.340 that that was,
00:41:58.120 and it's been 15 years,
00:41:59.580 so they're going to update
00:42:00.180 their recommendation
00:42:00.880 within the next couple years
00:42:02.020 whenever they have a joint meeting.
00:42:04.080 But that's definitely a vestige
00:42:05.800 of times past
00:42:06.600 when we thought that creatine
00:42:07.740 was harmful to healthy kidneys.
00:42:10.520 You just check us a statin C
00:42:11.860 because creatine makes
00:42:12.940 your creatinine blood marker
00:42:14.900 look abnormally high,
00:42:17.160 falsely high.
00:42:18.860 So creatine can make sense
00:42:20.400 in a lot of kids as well.
00:42:21.940 And then if there is a kid
00:42:23.460 that has really low insulin
00:42:24.500 and IGF-1,
00:42:25.320 sometimes Tomcat makes sense
00:42:26.940 in that individual.
00:42:28.700 And then in some kids
00:42:29.940 that do have optimal hormone profiles,
00:42:32.160 let's say there's an athlete
00:42:33.100 and he's developing
00:42:34.000 or she's developing
00:42:35.180 and they have very high testosterone,
00:42:37.660 very high IGF-1,
00:42:38.680 that's great.
00:42:39.480 You know that myostatin levels
00:42:40.760 are going to be really high
00:42:42.220 after you have that burst
00:42:44.320 of androgen during adolescence.
00:42:47.160 Myostatin is going to stop
00:42:48.560 the muscle from developing
00:42:49.640 and cause you to start putting
00:42:51.400 more fat into the tissue.
00:42:53.400 I think that myostatin inhibitors,
00:42:55.520 weak ones like fortitropin,
00:42:56.980 which comes from fertilized egg yolks
00:42:59.400 or epicatechin.
00:43:01.700 Cocovia is a good source
00:43:03.020 of epicatechins.
00:43:04.440 Different cocoa powders
00:43:05.500 have a lot of epicatechins.
00:43:06.660 Green tea has EGCG,
00:43:08.500 which is another epicatechin.
00:43:10.280 Basically, those take down
00:43:11.380 the levels of myostatin.
00:43:12.700 Those are also very reasonable
00:43:13.840 to take for the right patient.
00:43:15.760 What about should parents
00:43:16.600 be sweating about xenestrogens
00:43:18.340 in their kids?
00:43:19.120 Like, you know,
00:43:19.640 make sure they get certain types
00:43:20.500 of deodorants
00:43:21.280 for cosmetic products
00:43:22.320 and avoiding plastics.
00:43:24.920 Bisphenol A and phthalates.
00:43:27.240 Yes.
00:43:28.440 That's kind of where
00:43:29.420 I personally draw the line,
00:43:31.040 where if you are worried
00:43:33.220 about every single thing,
00:43:34.460 we live in an unnatural environment,
00:43:36.740 more so than ever.
00:43:38.520 So those are usually the ones
00:43:39.600 that I say to avoid.
00:43:40.600 If you live in an area
00:43:41.580 that more likely has contaminants
00:43:43.400 and microplastics,
00:43:44.920 a lot of times I do recommend
00:43:46.380 testing your water.
00:43:47.680 There are a lot of services
00:43:48.440 that do this.
00:43:49.300 I personally used my tap score
00:43:51.540 to test both the water
00:43:52.820 from the tap and the water
00:43:54.760 through my Berkey filter.
00:43:56.140 If you have young children,
00:43:57.980 then that seems like
00:43:58.760 a very reasonable time
00:43:59.900 to use a water filter
00:44:02.420 if you don't know
00:44:04.120 what the contents
00:44:05.220 of your water is.
00:44:06.460 And then as far as foods,
00:44:07.780 of course,
00:44:08.080 avoiding ultra-processed foods.
00:44:10.400 I think it was like
00:44:11.180 ultra-processed mac and cheese
00:44:12.600 that got a bad name
00:44:13.760 for having high phthalates.
00:44:14.940 I assume they fixed that by now,
00:44:16.300 but I actually don't know.
00:44:17.880 So a lot of times
00:44:19.060 it's the same recommendations
00:44:20.100 as any other whole food diet.
00:44:22.120 And then know your sources.
00:44:24.280 Try to avoid contaminants
00:44:26.240 at very high levels
00:44:27.120 and use the Pareto principle.
00:44:29.080 You know,
00:44:29.560 try to do right most of the time
00:44:31.420 and you'll get most of the benefit
00:44:32.700 even if you're just doing it
00:44:34.060 some of the time.
00:44:35.520 Well, Kyle,
00:44:35.760 this has been a great conversation.
00:44:37.000 Where can people go
00:44:37.500 to learn more about your work?
00:44:39.180 My help is on Instagram,
00:44:40.620 KyleGilletteMD,
00:44:42.020 and it's Gillette Health
00:44:43.040 on all other platforms.
00:44:44.800 I do have a podcast
00:44:45.600 that we fairly recently
00:44:47.280 have, I guess,
00:44:49.400 gotten pretty good audio
00:44:50.400 and video of,
00:44:51.160 but that's on YouTube,
00:44:52.100 Spotify,
00:44:52.640 and Apple Podcasts.
00:44:53.920 We have a clinically,
00:44:56.040 I guess,
00:44:56.460 a clinical-grade podcast
00:44:57.520 and then we have
00:44:58.320 a layman's podcast
00:44:59.860 that we're going to call
00:45:00.760 After Hours,
00:45:01.780 which should provide
00:45:03.060 good entertainment.
00:45:04.500 Fantastic.
00:45:04.880 Well, Dr. Kyle Gillette,
00:45:05.800 thanks for your time.
00:45:06.280 It's been a pleasure.
00:45:07.680 Thank you.
00:45:08.900 My guest today
00:45:09.540 was Dr. Kyle Gillette.
00:45:10.580 You can find more information
00:45:11.380 about his work
00:45:11.840 at his website,
00:45:12.760 GilletteHealth.com.
00:45:14.360 Also check out his podcast,
00:45:15.340 GilletteHealthPodcast
00:45:16.540 and check out our show notes
00:45:18.180 at AOM.IS
00:45:19.000 slash OptimalT
00:45:19.840 where you can find links
00:45:20.500 to resources
00:45:21.120 and we can delve deeper
00:45:21.780 into this topic.
00:45:29.820 Well, that wraps up
00:45:30.820 another edition
00:45:31.520 of the AOM Podcast.
00:45:32.620 Make sure to check out
00:45:33.280 our website
00:45:33.660 at ArtOfManliness.com
00:45:34.760 where you can find
00:45:35.040 our podcast archives
00:45:36.040 as well as thousands
00:45:37.100 of articles
00:45:37.480 that we've written
00:45:37.960 over the years
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