The Mikhaila Peterson Podcast - July 23, 2024


Harvard Neuroscientist on Gender Dysphoria, Intersex Conditions and Colleges | Adam Omary EP 206


Episode Stats


Length

1 hour and 26 minutes

Words per minute

162.72

Word count

14,114

Sentence count

557

Harmful content

Misogyny

21

sentences flagged

Toxicity

33

sentences flagged

Hate speech

42

sentences flagged


Transcript

Transcript generated with Whisper (turbo).
Misogyny classifications generated with MilaNLProc/bert-base-uncased-ear-misogyny .
Toxicity classifications generated with s-nlp/roberta_toxicity_classifier .
Hate speech classifications generated with facebook/roberta-hate-speech-dynabench-r4-target .
00:00:00.000 How did I actually get to researching hormones and brain development and end up with this as a
00:00:04.660 research focus? I didn't have friends. I was shy. I was homeschooled for two years in high school,
00:00:09.960 but that was only after being bullied a lot. Even something like eye contact, the way I'm
00:00:14.500 looking at you now, I used to be really afraid of that. I really only socialized and found joy
00:00:19.300 on online video games. Online video games, it's mostly males. The few girls that you'll see there,
00:00:27.160 They get tons of attention. I learned at a pretty early age. I can do that
00:00:31.480 I'd even catfish and like pretend to be someone's girlfriend. Your dad's Bible series
00:00:37.300 Resonated a lot with me. He really talks about the depths of those negative experiences
00:00:42.820 The fundamental question for a transgender identity is is it more similar to 0.57
00:00:48.280 Sexual orientation born this way or is it more similar to anorexia social contagion?
00:00:54.040 I think the answer is both for different people. That's the oversimplification. The nuance here
00:00:59.660 is that... Adam Omori, welcome to my podcast. Thank you, Michaela. Pleased to be here. Great.
00:01:09.740 Well, I'm really excited for this. This is going to be interesting, but I think we should jump
00:01:14.220 right in. We were talking, we went out for dinner last night and we were talking about
00:01:17.440 the famous twin study. So can you describe what that is? Dr. John Money was a famous psychologist
00:01:25.440 and sexologist. He was one of the first people to really promote sex change surgery for transgender
00:01:33.480 people and particularly for transgender kids. So this was happening in the 1950s and 60s and beyond.
00:01:40.260 And it was coming off of this blank slateist social constructivist view to gender. So we
00:01:46.560 didn't know as much about genetics or how sex hormones influence brain development during
00:01:52.900 critical windows prenatally are the first early signs of sex differentiation. So the theory was
00:01:59.860 in absence of all that and knowing how powerful behaviorism was, how powerful social learning is,
00:02:06.920 that all of these gender stereotypes that we see, and given that so many of them are culturally
00:02:11.700 specific, we don't know if you were to raise a child of one sex completely as the opposite sex,
00:02:19.020 everything, everyone was on board, and even their body, like if you did a sex reassignment surgery,
00:02:24.560 if this genetic male child had a vagina and was raised as a girl all his or her life, 0.65
00:02:31.980 then would they identify as a healthy, functional woman? And it was an open question, but there was 0.99
00:02:40.700 enough of a theory to think that that's very well what will happen so there have been various cases
00:02:50.100 but the ideal would be a twin study like people with the exact same genetics exact same environment
00:02:57.120 and you take one and raise them the opposite sex but everyone knew it would be unethical
00:03:03.200 so it was pretty much a fluke that this perfect natural experiment came along a set of twin boys 0.81
00:03:10.680 born in Canada, they had a circumcision and one of the circumcisions was botched and the 0.99
00:03:16.560 boy's penis was destroyed. And because of this, they were referred to all sorts of medical 0.67
00:03:24.060 specialists, including John Money. And the theory was this was also based on Freudian
00:03:29.580 psychoanalytic influence, the thinking about sexual development being critical for healthy
00:03:35.500 psychological development. So this thought of this boy growing up without a penis, he's going to be 0.99
00:03:41.900 very insecure in his manhood. He's never going to have a functional sexual life. How's he going to 0.99
00:03:47.160 get married? He's going to be depressed. He'd be way better off living as a woman. We can construct
00:03:53.340 a functional vagina. At the time, vaginoplasties had been developed, but it's much harder to 0.97
00:03:59.780 construct an artificial penis, especially in a child. So the thought was, here's this perfect 0.89
00:04:05.600 natural experiment. So part of it, I think, was this authentic desire to promote what they thought
00:04:13.180 would be in the child's best interest. But the other part of it that certainly was in the back
00:04:18.780 of John Money's mind is this is the perfect experiment to test my theory. Yeah, that's
00:04:23.700 horrifying so this child was raised as a girl brenda and her brother brian was a typical healthy
00:04:33.260 boy and brenda was very tomboyish and at early ages like before eight or so experienced what
00:04:43.460 we would now call gender dysphoria but didn't really know how to say it other than i don't
00:04:49.580 feel like other girls but not just tomboyish was the kid told what had happened or did eventually
00:04:56.440 eventually but not at that time not till he was 14 wow so he was tomboyish but not just that you
00:05:04.540 know because he was told well lots of girls are kind of more rough and tumble they're into boys
00:05:09.100 things but he felt no it's not just that like i don't feel like i'm in the right body and again
00:05:15.620 without really having the language for that and he's told this to his doctors but john money turned
00:05:21.760 out to be quite biased so in all of the papers that were published at the time in that first
00:05:26.840 decade or two you hear nothing but a success story of look there's twins he was born a male
00:05:33.420 now he's living as a healthy girl so feminine you wouldn't believe it wears dresses says he's a girl
00:05:38.740 everything like that wow but experiencing gender dysphoria and depression and anxiety symptoms 0.58
00:05:45.900 the entire time and then once the puberty age comes along well it's not a girl so he's not
00:05:53.360 going to naturally go through puberty so there's this critical juncture where you have to start 0.66
00:05:59.140 taking estrogen and they did give him estrogen for a brief time and started growing breasts
00:06:06.520 and then combined with the depression that she was already experiencing and you see the pronouns
00:06:12.580 are confusing like i'm already going a bit back and forth between he and she so this case isn't 1.00
00:06:18.280 working and so she gains a lot of weight in order to hide her breasts because she hates this female
00:06:23.980 figure that the estrogen is developing and just starts to become suicidal so finally after all
00:06:32.180 that and the trauma that it causes in this family the parents tell her tell him what happened and
00:06:38.380 he said suddenly it all clicked that I was born a boy this is why I'm so much like my brother
00:06:44.780 it's not just that we're opposite sex twins and we share some interests like literally we share
00:06:50.100 all of our genes I was meant to be a boy and then you might think okay so gender identity
00:06:57.160 is genetically ingrained. And actually I should finish telling you about this story. So it's not
00:07:06.280 a happy ending. Oh no. So Brenda, after 14 or so, decides to live as a boy, stops taking the
00:07:16.360 estrogen, starts taking testosterone, even gets surgery to construct a penis later and renames 0.51
00:07:22.980 himself David, David Reimer, and continues living as a man for another two decades or so, 0.50
00:07:29.560 even gets married and has kids, but tragically ends in suicide. He struggled with depression
00:07:36.680 his whole life. John Money, the sexologist, while doing these yearly check-ins that he would do with
00:07:43.720 the twins, he'd show them porn. He'd ask them to strip down and do all sorts of weird tests. He was 0.69
00:07:51.660 in this mindset that america is so puritan and that we're so repressive in our attitude towards
00:08:02.400 sex and we just need to liberate it so for example he thought that homosexuality which at the time
00:08:08.360 was still in the dsm as a mental disorder he thought that it was a matter of children not
00:08:14.040 being exposed to healthy sexual role models so he also encouraged david and brian's parents to 0.93
00:08:22.120 walk around nude and have sex in front of the children they didn't do that they drew the line
00:08:26.960 there but his theory and when brenda entered puberty so she's still a girl before she knew 0.60
00:08:34.240 what happened she thought she was gay she was attracted to other girls so that's interesting
00:08:39.600 genetically is there something that sexuality encodes whether it's you're homosexual or straight
00:08:47.940 it's completely dissociable from gender identity so the twin study showed that as well and john
00:08:54.080 money you know he was he was willing to admit that part show that in some cases she's more tomboyish
00:09:00.460 maybe she's gay but still she's what we would now call cisgender it was a success in that regard
00:09:06.440 but he was so adamant that it must have been because the parents had this unhealthy attitude
00:09:13.720 towards sex the parents marriage really was falling apart ever since this botched circumcision
00:09:19.720 ruined their family so john money's theory was brenda's gay not because she was born a boy 0.90
00:09:25.740 but because she witnessed this unhealthy marital dynamic oh yeah of course blame the parents rather
00:09:31.100 than thinking maybe, maybe he could be wrong. Blame the parents. Yeah. Right. So this makes
00:09:36.220 us think, okay, now with modern knowledge of genetics, gender identity, and maybe sexual
00:09:41.400 orientation, it must be genetic. And then in grad school, I learned about this intersex condition,
00:09:47.820 complete androgen insensitivity syndrome. So the most extreme case of this with complete androgen
00:09:54.200 insensitivity, that means that you can produce androgens like testosterone, but the androgen
00:10:00.260 receptor doesn't work, so it's functionally inert. So you can have a genetic male with a Y-chromosome,
00:10:07.540 and I didn't learn this until grad school either. The Y-chromosome, what it really does, the SRY
00:10:13.680 gene on the Y-chromosome produces testes determining factor, which is a protein that's
00:10:19.660 used to construct the testes out of undifferentiated gonadal cells. So in the first six
00:10:26.060 weeks of fetal development. The gonads are sexually undifferentiated. And I initially
00:10:31.740 assumed that it was the Y chromosome that coded for development of the penis and other secondary
00:10:37.060 sex characteristics. No, that's not true. It's all downstream effects of testosterone. The only
00:10:43.400 thing the Y chromosome does is it determines whether you develop testes or an absence of a
00:10:48.420 Y chromosome ovaries during that early period of fetal development. That's interesting. Okay. So
00:10:54.500 in a genetic male with complete androgen insensitivity, first six weeks of fetal
00:10:59.960 development look normal. And then they do produce testes, and the testes do produce testosterone,
00:11:06.280 but the androgen receptor doesn't work. So it doesn't bind to anything. So functionally,
00:11:11.360 it's inert. And free-floating testosterone in males can interact with an enzyme called
00:11:17.360 aromatase. So it's aromatized into estrogen. And those low levels of estrogen that are typical
00:11:24.300 in males are enough for the development of female characteristics. It's normally inhibited by
00:11:30.640 testosterone, but in absence of any testosterone, even if you have a Y chromosome, the baby will 0.78
00:11:37.700 develop a vagina and be assigned female at birth. So that's interesting too, because people
00:11:43.620 criticize the binary sex idea and say, it's a social construct. It's assigned at birth. It's
00:11:50.080 an arbitrary physician rating. Obviously, it's not arbitrary. In 99.9% of cases, there's complete
00:11:56.580 overlap between genetic sex, phenotypic sex based on external genitalia, hormonal sex. But in some
00:12:04.020 of these intersex disorders, they are dissociable. So you can have a genetic male identified female
00:12:10.140 at birth and then raised as a girl. So this is in a way a more perfect case than the twin study,
00:12:16.280 because in the twin study everyone knew that at least for the first couple weeks or months up to
00:12:21.840 the circumcision this was a boy and even the social constructivist could counter well they
00:12:27.780 weren't brenda wasn't really raised as a girl because everyone knew in the back of their mind
00:12:31.660 that she's secretly a boy so maybe she's tomboyish because they were treating her different in these
00:12:37.080 unconscious ways yeah but with cais they're genetically male they're raised as female 0.80
00:12:42.820 they don't even know anything's wrong with them because again perfectly healthy female genitalia 1.00
00:12:47.920 so then you might think they're going to live their lives as prepubescent girls because they 1.00
00:12:54.020 don't have any testosterone they're not going to go through male puberty but they don't have 0.99
00:12:57.500 ovaries how are they going to go through female puberty but it turns out that low level of
00:13:02.800 estrogen and progesterone that males have is enough for the development of female secondary
00:13:07.900 sex characteristics. Wow. It just normally is inhibited by testosterone. So then these girls 0.96
00:13:13.100 will start growing breasts, fat will redistribute around the hips, other female secondary sex 1.00
00:13:18.340 characteristics, but they won't ever get their period because they don't have ovaries. Wow. So 1.00
00:13:23.560 a girl with CAIS, even though they're genetically male here, I'm using the term girl. And I think 0.67
00:13:29.420 that would be appropriate. They're going to identify as girls. They're even overwhelmingly
00:13:34.000 traditionally attracted to men, exclusively heterosexual, and identify as women. 0.99
00:13:41.720 But they are essentially infertile women, and they don't find out until they're 15, 16, 0.99
00:13:47.260 have never gotten a period. They go to the doctor to see what's wrong, and they find out that they 0.77
00:13:52.620 don't have ovaries. They have testes inside their abdomen that never descended. That's crazy.
00:13:58.160 What percentage of the population does this hit? Complete androgen insensitivity,
00:14:02.900 definitely less than one in 10 000 yeah okay okay that's still quite a few that would be shocking
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00:14:57.240 All right. So what that tells us, it's not genetics alone. It's prenatal hormones during
00:15:03.080 a specific critical window of brain development that seems to determine gender identity and
00:15:10.420 sexual orientation. And there's a couple other intersex disorders that we can use to hone in
00:15:15.200 on that hypothesis further. All right. So another one is congenital adrenal hyperplasia. This is
00:15:22.360 one that I've published original research on. So congenital means present from birth. Adrenal
00:15:28.300 hyperplasia means overactive adrenal glands. So what happens due to a genetic disorder that
00:15:35.420 results in deficiency in an enzyme called 21-hydroxylase, which is an enzyme necessary
00:15:42.400 to synthesize cortisol, the adrenals go into overdrive, attempting to produce more cortisol.
00:15:50.180 and cortisol, everyone knows, is a stress hormone, but it's also necessary in low levels just for
00:15:56.500 basic functioning like metabolism. So adrenals go into overdrive. They begin producing other
00:16:03.120 precursor androgens that can be synthesized into cortisol typically, but can also be synthesized
00:16:10.060 into testosterone. And since their cortisol system doesn't work properly, they have excess levels of
00:16:16.140 testosterone. And CAH can affect both males and females. But if it happens to a male, well,
00:16:22.680 they'll have more testosterone, but it's within the male typical range of variability, which is
00:16:27.020 very large. And it can be deadly if left untreated, but typically it's diagnosed shortly after birth
00:16:33.180 and they're given glucocorticoid supplementation. So the real effects happen in females because then 0.91
00:16:39.240 they have male typical levels of prenatal testosterone during this critical window for 0.97
00:16:44.280 brain development. So there's been decades of studies of girls with CAH and they turn out
00:16:50.340 overwhelmingly to be more tomboyish, more interested in rough and tumble play exhibit
00:16:56.280 boy play preferences. And during adulthood, 30 to 40% of them compared to about one to 2%
00:17:03.380 in the general population identify as gay or bisexual. Wow. So those two pieces put together
00:17:10.960 CAIS and CAH to me are very strong evidence that it's prenatal sex hormones and particularly
00:17:18.680 androgen exposure that determine whether in absence of androgens, you're going to develop
00:17:23.900 a female phenotype and be attracted to men. And in presence of androgens, you're at the very least
00:17:30.860 more likely to be attracted to women. Like the girls with CAH, they're not overwhelmingly gay.
00:17:37.480 in fact a majority of them aren't but way more likely to be and then similarly for males most
00:17:45.220 males are attracted to women and yet genetic males without any testosterone are overwhelmingly
00:17:50.860 attracted to men that's interesting wow that is interesting okay so you know a lot about this
00:17:57.780 there's one more interesting okay go keep going no no keep going okay i should also add cah is
00:18:04.560 the most common, and it affects about one out of every 10,000 births. So I think that the other
00:18:10.180 ones being more rare than that, add them all up, and you're probably looking at less than one in
00:18:15.560 a thousand for all of these severe intersex conditions. I should also add the term intersex,
00:18:22.320 some people now say is outdated or offensive. The technical term that people like is differences in
00:18:29.280 sexual development or DSDs. I think intersex is more straightforward, and we're talking to a
00:18:34.080 general audience here, so that's an important distinction to make. But 5-alpha reductase
00:18:40.680 deficiency. So earlier I said it's all testosterone that determines whether you grow a penis and it's 0.92
00:18:46.920 testosterone that acts on the brain to produce these psychological sex differences. That's the 0.76
00:18:52.440 oversimplification. The nuance here is that testosterone interacts with an enzyme called
00:18:58.760 5-alpha reductase, and it's converted into dihydrotestosterone, DHT. And DHT is what
00:19:06.380 actually constructs the penis. So with 5-alpha reductase deficiency, this is another really 0.79
00:19:12.420 interesting natural experiment where you can have a genetic male with typical levels of testosterone
00:19:18.860 and functioning androgen receptors. So testosterone organizes the brain in the male typical way,
00:19:24.740 but they don't have any DHT so they're not going to be born with a penis and again in absence of 0.99
00:19:30.620 that female typical development is the blank slate default condition so they'll be born with a vagina 0.98
00:19:37.280 they'll be identified female at birth they'll probably be more tomboyish like CAH although I 0.82
00:19:43.020 don't know of any evidence on that this condition is much rarer and then at puberty this is where
00:19:49.760 it really gets different because they still have testes and the testes still produce testosterone 0.78
00:19:55.400 they have a vagina but the testosterone is what produces other secondary sex characteristics 0.99
00:20:03.240 so then you have this girl who's maybe more tomboyish maybe not but has lived her life 0.98
00:20:08.680 identifying as a girl and then suddenly she hits puberty and starts growing a beard starts deepening
00:20:14.260 her voice, other male characteristics. And this one is really interesting because the other
00:20:19.520 conditions I told you about, the gender dysphoria tends to manifest. Well, with CAIS, there's not
00:20:25.600 even any dysphoria. They're just completely woman. They find out they're infertile, but other than
00:20:30.100 that, they're healthy. With CAH, they're more likely to experience gender dysphoria. And that's 0.56
00:20:35.180 pretty much a function of how much prenatal androgens they're exposed to, because in the most
00:20:40.880 severe form the testosterone will even masculinize their genitalia so it'll be full-on intersex 0.74
00:20:48.020 ambiguous genitalia the clitoris will grow into what looks like a micropenis the labia will grow
00:20:54.960 outwards and if it's severe enough even fuse into a scrotum but not fully functional you'll also see
00:21:03.040 this with partial androgen insensitivity so the complete condition is the most straightforward
00:21:08.080 it's also the rarest. But if you have partial androgen insensitivity with lower than average
00:21:15.240 testosterone, it could go anywhere from having, say, a clinically underdeveloped micropenis 0.94
00:21:21.680 to ambiguous genitalia if you have, say, not enough to grow a functional penis, but also way 0.96
00:21:28.460 more than female typical. So when people say sex is a spectrum, sex itself is binary, but just the 0.90
00:21:36.340 forms that your genitals can take during early development do take on this spectrum that's
00:21:42.480 dependent on how much prenatal testosterone or particularly DHT you're exposed to. Okay,
00:21:48.740 so back to 5-alpha reductase. Without any DHT and with testosterone and with secondary male
00:21:56.720 sex characteristics at puberty, it's about a 50-50 shot. Some of them experience gender dysphoria
00:22:04.160 and want to go on androgen blockers and continue to live in their lives as women. 0.82
00:22:09.260 They take estrogen to grow the female characteristics that their body can't naturally produce 0.99
00:22:13.780 and go on to live healthy lives as unfertile women. 0.81
00:22:18.260 And others decide to fully let the testosterone take its course,
00:22:23.020 and maybe they can get a phalloplasty to continue to live their lives as men.
00:22:28.740 That's crazy.
00:22:30.000 And you said that, so all of that is one in a thousand people, you think?
00:22:34.160 I would say one in a thousand would be the umbrella stat for any one of these different
00:22:40.160 disorders and even that is a massive overestimate because CAH for example even though it's classified
00:22:47.220 as an intersex condition well for one thing in half of them it happens in males so you're going
00:22:53.920 to experience the same medical issues with cortisol deficiency but no real issues of sex
00:23:01.500 development. And then even of the females who have CAH, it's only ambiguous genitalia and gender 0.98
00:23:09.500 dysphoria in the most extreme cases. Maybe they'll turn out to be gay, but they can still 1.00
00:23:13.900 live perfectly healthy lives as women and they are genetically women. So that's wild. Okay. So 0.96
00:23:22.300 a few questions. One, why do you know so much about this? Like what was it about my whole PhD?
00:23:28.200 Yeah, but what made you decide to study this? Was it like rampant talk about gender dysphoria that you grew up around? Or like, why did this interest you specifically? It's fascinating. You do a lecture on Peterson Academy, probably. That sounded like a science class. That was really good. Yeah, that was really good. But what made you decide to become interested in this?
00:23:49.880 thank you Michaela well there's two separate answers there's the practical how did I actually
00:23:56.420 get to researching hormones and brain development and end up with this as a research focus and then
00:24:01.780 there's like you said what makes me interested in it because it sounds so niche right so this
00:24:07.640 actually connects to I think some of the personal interests that we're going to get into like about
00:24:14.340 my upbringing being homeschooled and growing up with a single mom and spending a lot of time
00:24:22.760 online and having to learn social dynamics in a somewhat unorthodox way. But I'll start with
00:24:30.000 the research that's more straightforward. So I came into college actually to study physics,
00:24:35.400 but pretty quickly I switched into cognitive science. At first, stemming from the physics
00:24:40.220 background. I was interested in artificial intelligence and thinking I was going to do
00:24:44.280 something very computer programming. I was good at math. But I realized pretty early on in these
00:24:51.680 classes that we know way less about human intelligence. How are we going to build artificial
00:24:58.380 intelligence? I mean, we have succeeded in a way, but we also can't really debate on, you know,
00:25:04.380 what is a woman? So to me, as you mentioned, some of this political stuff going on in the
00:25:10.920 background, it made me think there were more foundational philosophical questions to be
00:25:15.220 addressed through neuroscience. And then my first neuroscience lab, really just by happenstance,
00:25:22.520 happened to be focusing on hormones and brain development. And I got to work with CAH populations.
00:25:29.040 So this was pre-collected data, and we were looking at structural brain differences between children with and without CAH.
00:25:37.620 So the other thing that's interesting about this natural experiment design, you can test two things simultaneously about the hormones.
00:25:44.280 So if you see an effect that's present in both males and females with CAH, but not in typically developing children,
00:25:51.420 then it's probably a result of either the cortisol deficiency prenatally or more likely the glucocorticoid supplementation.
00:25:59.040 Because if you're taking chronic low levels of cortisol, you know, it's almost like chronic stress exposure, which we know impacts the brain, hippocampal volume in particular, which is the biggest effect that we found.
00:26:10.880 But if you see an effect that's only present in CAH females relative to control females, then it's probably the prenatal androgen exposure.
00:26:21.500 And you do see both of those.
00:26:23.320 You see some effects that are probably glucocorticoid driven present in both sexes.
00:26:27.940 and then you see some effects, including the behavioral and psychological ones that are only
00:26:33.060 present in females. Interesting. Okay. Before we get into your background, what does, what does
00:26:39.640 cortisol, what does, what do hyper stressful situations like long-term do to the brain?
00:26:44.560 Oh, this isn't my area of expertise. Robert Sapolsky has a great book on this called
00:26:49.040 why zebras don't get ulcers. And the basic idea, I guess there's two. The first is very low levels
00:26:55.760 of cortisol, it has other physiological functions that are just necessary for survival. But I'm not
00:27:01.480 a physician. I couldn't list those out. But then in terms of psychological stress response, this
00:27:06.540 fight or flight response, we know that cortisol, a spike in the short term, is really adaptive.
00:27:13.020 So why don't zebras get ulcers? Well, they'll experience a massive cortisol spike when they're
00:27:18.120 running away from a lion. And then, you know, they chill out. Only humans, actually primates,
00:27:23.920 Sapolsky's done great work on this. We'll experience chronic stress, even from social
00:27:28.120 bullying, like anything, even if all of your physiological needs are met, we have social
00:27:33.840 needs too as social mammals. And what does chronic stress do? Can you reverse it? I know, I know this
00:27:40.160 isn't your area of expertise, but does it shrink your brain? It can shrink your hippocampus, which
00:27:45.420 is necessary for forming memories. It can impair memory, especially if this happens during early
00:27:52.000 development so there's this idea of sensitive or critical windows for brain development a critical
00:27:58.220 is like fully lose it or use it use it or lose it and you'll see so a lot of these early studies
00:28:06.220 they sound very mad scientisty and they kind of are so for whatever reason a lot of the early
00:28:12.220 studies on visual development were done on kittens and you know when you say we're done
00:28:19.380 in terms of a neuroscience experiment, often it ends with the animal being euthanized because
00:28:24.900 you have to dissect its brain. So rodents are most commonly used for these invasive neuroscience
00:28:32.540 studies, but cats, maybe because they were larger, maybe because they have a longer window of
00:28:39.500 plasticity. So plasticity is the term for how malleable your brain is and during specific
00:28:44.820 windows. So you'll see that there's sort of this parallel between evolution and ontogeny development
00:28:53.280 within a particular lifespan. So the idea is the earlier a function is, like sensory motor brain
00:29:00.020 regions mature the earliest, like within the first few months. So these scientists, what they would
00:29:05.680 do, they would blindfold kittens for the first few months of their life. And if they did that,
00:29:11.400 and they had no light exposure, then they would be permanently blind into adulthood. But if you
00:29:17.580 took a kitten who was exposed to normal light and developed normal vision for the first few months,
00:29:23.900 and then you blindfold them after the visual cortex matures, and then you blindfold them for
00:29:29.960 years, they'll still be able to see when you take it off. So that's the strongest example I know of
00:29:35.800 for critical period. And then psychologists have taken this for language. There's also a pretty
00:29:41.540 strong critical period for language development in the first five or so years of life. And then
00:29:46.540 in adolescence, in my main area of study, there's argument that it's a critical period for higher
00:29:55.520 order cognitive development. So this is things like planning about the future, evaluating risks,
00:30:01.480 social cognition emotion regulation high level stuff like that but then there's also this term
00:30:07.060 critical versus sensitive so with vision in the first few months if you don't have light exposure
00:30:13.300 no amount of light exposure later on will help for sensitive period it's more like it's a period
00:30:20.400 of heightened plasticity but it's not all or nothing wow that's crazy okay you might not know
00:30:28.240 anything about this but i'm going to ask you anyway so if you put people on something like
00:30:32.520 and this this pertains to me because i was on i was on medication throughout my developmental years
00:30:36.920 i've always wondered what that did to my brain and i'm not saying you know that because i don't
00:30:41.940 know if anybody knows that but if you put somebody what's your guess here if you put a kid on
00:30:47.740 something like an ssri because like my experience was it dampened down i like i took risks when i
00:30:54.840 shouldn't have taken risks and that's part of my personality but i don't know how much that was
00:30:58.880 influenced by the medication do you have any thoughts here if you become more of a risk prone
00:31:05.060 personality type or even more disagreeable but it's because of early serotonin levels i don't
00:31:10.620 know i wonder okay because i like i know i would put myself into risky situations without being
00:31:15.920 scared and i'm sure that that that was blunted from the ssris so i've always just wondered
00:31:20.820 throughout my developmental years if that made me more less risk averse than i should have been
00:31:25.360 not that i'm like unhappy about it or anything but it was like maybe so there's there is a tie
00:31:31.140 in there about other forms of medication that we give to kids so puberty blockers is the one
00:31:35.860 on the forefront of my mind and that's been an active area of research for me recently not working
00:31:41.360 with primary data but just reading animal studies giving them puberty blockers and looking at effects
00:31:46.780 of brain development and whether or not it's generalizable to humans, looking at the limited
00:31:51.140 number of human studies that have been published. But there is a meaningful connection there to
00:31:55.860 SSRIs, which is that a lot of people criticize the animal models of puberty blockers or hormone
00:32:02.220 replacement. They'll say it's not generalizable to transgender people or even just to humans
00:32:07.240 because, well, it's an animal model. But most research, most things we know about the brain
00:32:13.460 stems from animal research. And a lot of these things, especially subcortical structures,
00:32:19.580 the more basic, the earlier evolved the brain region is, the more likely it is to be conserved
00:32:25.040 across mammals. And mammalian brains are more similar than they are different. They operate
00:32:30.580 on the same neurotransmitters, the same hormones, same sensitive periods during phases of life.
00:32:36.860 And you'll have these behavioral markers that you'll use to test whether a drug works or not.
00:32:42.080 So something like antidepressants, you're not just going to test it on a human right away.
00:32:46.860 You want to have a rodent model for it first, but how are you going to measure whether a rodent is depressed or whether it's actually alleviating depression?
00:32:54.460 And I don't know the specifics, but the basic idea is they have a whole array of standardized paradigms looking at things like appetite or play behavior or sex behavior.
00:33:05.260 and researchers have coded how a typical animal responds and how a depressed-like animal responds
00:33:12.840 or an anxious-like animal responds. And generally, you'll validate whether an antidepressant or an
00:33:17.760 anti-anxiety med works by looking at the behavior. So the interesting thing is, in these puberty
00:33:23.060 blocker studies, there's some preliminary evidence in rodents and in sheep that they may increase
00:33:30.920 anxiety or depressive-like symptoms. Now, we don't know if that generalizes to humans,
00:33:36.900 but based on the psychopharmacology of how antidepressants and anti-anxiety meds work,
00:33:42.080 and just on this general theory that sex hormones are critical for brain development during puberty,
00:33:49.740 I'm sure that there's some effect. And in fact, we do know there's some effect. We just really
00:33:54.580 don't know what it's going to be. Like if you find a structural brain difference, with some,
00:34:00.040 it's really straightforward your hippocampus is shrinking you're going to have impaired memory
00:34:04.760 but for a lot of these especially for cortical networks where it there's enough plasticity
00:34:10.620 that there can be trade-offs so like one one area shrinks another area grows to take its place
00:34:17.080 like in the extreme examples of this you'll see that you know if a child loses one sensory system
00:34:23.280 at an early enough age other sensory systems will grow and improve to make up for it that's
00:34:28.640 neuroplasticity in action yeah that's really cool that's so that's what happens when somebody goes
00:34:33.220 blind really little right does their hearing actually improve i don't know if the hearing
00:34:37.540 itself improves i would think yes but at the very least we do know well depending on the type of
00:34:43.240 brain damage if it's literally brain damage like the area is lesioned and there's tissue that just
00:34:49.100 simply isn't there anymore then yeah auditory cortex will grow to fill in what used to be visual
00:34:54.860 cortex and presumably that comes with improved function oh wow that's cool if you guys are not
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00:36:27.300 the rest of this episode. Okay. Okay. Let's get into your background a little bit because
00:36:31.060 from what you told me yesterday and from what I know about you, it's interesting. And I think my
00:36:35.540 audience will find it interesting, but you grew up in LA, you had a single mom. What was your
00:36:41.400 childhood like? I played a lot of video games and my mom is a part-time piano teacher, music
00:36:50.000 director at her church we also wanted to talk about spirituality so that may tie in here but
00:36:55.780 basically I didn't have a strong male role model in my life I didn't have friends I was shy I was
00:37:04.340 homeschooled for two years in high school but that was only after being bullied a lot and I was
00:37:09.260 bullied for my weight I gained a lot of weight I was 250 pounds when I was 15 years old and
00:37:16.080 didn't have any friends didn't have any romantic interest I really only socialized and found joy
00:37:22.580 on online video games and it's such a different world and I think this is in a way where the
00:37:28.660 early obsession with sex differences comes in because online video games it's mostly males
00:37:34.520 and the few girls that you'll see there they get tons of attention often it looks like harassment
00:37:41.320 but on these video games and as a naive kid i was thinking you know there's this term on my favorite
00:37:48.000 game runescape buying gf buying girlfriend so people would literally say that and you know
00:37:53.700 sometimes you'd have someone trade them like they'd have this female character following them
00:37:58.400 around like entourage they'd give them money or free stuff so i learned at a pretty early age i
00:38:03.380 can do that i can play as a female character i'd even catfish and like pretend to be someone's
00:38:09.660 girlfriend. This might have been as early as 10 years old. I don't think I even knew what it meant
00:38:14.420 other than, hey, this person's going to give me free stuff. So I'd walk around as a female
00:38:19.240 character. I'd also go on these online chat rooms like Omegle or Kick Friends. And this was really
00:38:25.800 if I was lonely and trying to chat with people. So often what I'd find is I'll make a bio about
00:38:31.680 myself. I'll post about my interests. I'll say I'm 15 years old. I want to chat. I'd get no messages.
00:38:36.920 and then I make the exact same bio I say I'm a girl I get dozens of messages and a lot of them
00:38:45.380 are probably other lonely boys like me and we have common interests in video games they're like
00:38:50.480 it's so cool that you're into video games and you're a chick you're like the perfect bro you
00:38:55.160 know we bond but they they'd fall in love with me often a friendship like that would end because
00:39:00.640 they're like oh my gosh you're the perfect girl it's just like it's like you're a guy but you're
00:39:05.060 not. And often, I'd find that I could get more attention if I just put a pretty picture. So I'd
00:39:10.620 find random girls on the internet, and just post a random photo and pretend to be that person.
00:39:17.640 That never backfired, hopefully. So I'd also get lots of dick pics. And for the most part, 0.99
00:39:28.480 I just block them. The unfortunate part of it all. Right. But, you know, this was in the early
00:39:34.680 wild west of the internet at least for teenagers so this is back when you go in xbox live chat rooms
00:39:41.880 and everyone is cursing up a storm and you're sending like the weirdest porn you can find 0.94
00:39:48.460 or isis beheading videos like all of my video game friends were really fucked up and so was i so 0.97
00:39:55.180 i think part of that had to do with whatever almost unconscious resentment for not having 0.94
00:40:02.300 friends or being popular in the real world. So it's like, you know, part of it stemmed into this
00:40:07.260 early version of perfectionism. How can I get the highest level, get the best gear, you know,
00:40:13.840 form a clan and get into a high rank, like social status within this virtual world?
00:40:19.680 Eventually in my late teens, it snapped like I could just grind in real life, grind as if it's
00:40:26.160 a video game, work on my fitness, obsessively calorie count. I did all that and succeeded
00:40:31.780 after basically treating it like a video game. Yeah. Yeah. That was, that was wise. I think
00:40:39.660 people have to realize that my dad has talked a lot about, you know, people who, this is mostly
00:40:44.880 people who take drugs or something like that is you have to turn your life into the gamble, kind
00:40:49.560 of make it into the game. You have to have like enough going on. So you ended up, I want to get
00:40:54.960 into that you ended up losing a bunch of weight young which was very disciplined that's very
00:40:59.860 difficult for people to do especially the way you did it because you just you calorie count I didn't
00:41:04.800 do it smart though I calorie counted obsessively but I would eat the unhealthy food if it was lower
00:41:10.500 health if it was lower calories I didn't have really any knowledge of whole foods or like
00:41:15.440 what's healthy yeah still that's impressive though how old were you I was 16 when I started
00:41:23.180 I lost 80 pounds in 14 months. So I said I used to be 70 pounds heavier, but I got really skinny
00:41:31.300 first. I went all the way down to 165. And by then I was still shy and awkward, but I did have
00:41:40.060 this fantasy of, okay, I don't want to just graduate the weird homeschooled kid and never
00:41:45.440 experience graduation or prom or anything like that. So I wanted to get back into school. So
00:41:50.300 senior year I re-enrolled into public school I still didn't make friends I was still really shy
00:41:55.600 but I slowly started coming out of my shell more and that's when I was 165 pounds and then
00:42:01.280 senior year I started working out for the first time so by college I was back up to like 180
00:42:08.180 pounds and had some muscle less than I have now but like I looked normal and I only later learned 0.99
00:42:14.620 you know I graduated high school still without my first kiss but I later learned oh some girls
00:42:19.580 actually had a crush on me then they just spoke to me and I was so awkward that they lost it but
00:42:24.640 at the beginning it was like hey this is a new kid coming in he's not that bad looking he seems
00:42:29.580 to be smart and then he's practically autistic so you lose the interest there and then I get to 0.58
00:42:35.780 college and dating apps are a big thing so I get on tinder and it was still the same thing like I 0.99
00:42:42.120 started getting a good amount of matches I still had all my friends the ones that lasted were these
00:42:47.980 online friends that I'd never met in person from video games, a lot of them were still overweight
00:42:52.220 and awkward and still never had any success with girls. So just the fact that I was now reasonably
00:42:57.540 fit and going to USC, which has a very attractive student body and is a pretty social school.
00:43:05.220 It also has a great research department and it's connected with Children's Hospital of Los Angeles.
00:43:10.180 So I kind of just steered into the research direction after I failed to succeed at the
00:43:16.540 extroverted party life. But online apps were different because, well, at first you begin
00:43:24.300 texting and I was familiar with that, right? Not only that, but in some ways, I think I got more
00:43:31.540 in touch with my anima. We can use the Jungian term, like my feminine characteristics, because
00:43:37.920 I spent so long pretending to be a girl and, you know, not wanting to be completely unrealistic.
00:43:44.380 I was telling Rob about this, Rob Henderson, and we use this term authentic catfish.
00:43:50.360 He'd be like, that's a good title for an article.
00:43:52.980 That's a good what's an oxymoron, right?
00:43:56.460 So I'd have these one on one dates.
00:43:59.600 I did reasonably well at scheduling dates because, again, it's all texting.
00:44:04.100 And then they'd meet me first five minutes, see how awkward I am, find an excuse to leave.
00:44:09.820 And I slowly got better through trial and error.
00:44:11.940 but I still didn't know how to interact with people like in class or in a real setting or at
00:44:17.120 parties. I didn't even get invited to parties. It slowly got better. And, you know, I had my first
00:44:22.940 few relationships and became more outgoing, like even something like eye contact, the way I'm
00:44:30.000 looking at you now, I used to be really afraid of that. And your dad was a big help. Red pill
00:44:35.200 community in its own way. It was a big help, at least insofar as it got me on the stepping stone. 1.00
00:44:41.940 to reading and listening to better male role models because red pill online it's almost like
00:44:48.820 a spin-off of the incel community right it's like the incels that and i was like that right
00:44:54.880 involuntary celibate graduated high school without a first kiss overweight and then lanky and awkward
00:45:00.940 even after he loses the weight but it slowly got better and the red pill people it there was a lot
00:45:09.240 of misogyny there it's almost like they adopted this self-mind this self-improvement mindset but
00:45:14.660 then simultaneously had this deep insecurity like if it didn't work then it's the girl's fault
00:45:20.640 because and they're you know they're hyping each other up and some of it is genuinely positive you
00:45:25.440 know they're saying here's how to count calories here's how to go to the gym here's what you should
00:45:29.760 do look people in the eye give them a firm handshake like all of that stuff really helped me
00:45:33.760 but and i'm pretty sure it was from there red pill on reddit that i stumbled across
00:45:39.860 one of your dad's first videos and then i was hooked that also was a major contributor to me
00:45:45.420 switching to cognitive science from physics getting interested in psychology and brain development
00:45:50.680 and then phd in the department he used to work at the rest is history yeah so are you in david
00:45:57.920 buss's lab david buss is at ut austin and he's an evolutionary psychologist whose lab are you in
00:46:03.660 I'm in Leah Somerville's lab, and she specializes in adolescent brain development.
00:46:09.060 But the puberty and hormones research is also fairly new to our lab.
00:46:13.320 We have this NIH grant that we're working on.
00:46:16.420 This is all part of the Human Connectome Project, which is a massive study.
00:46:20.840 There are different wings of it.
00:46:22.800 So there's the main Human Connectome Project, which is brain development in adults ages 22 to 65.
00:46:30.120 there's a separate cohort looking at brain aging 65 plus there's a infancy and toddler cohort and
00:46:38.800 then there's the development one which we're working on which is 1300 kids ages 5 to 21 and
00:46:46.320 you have different functional MRI measures structural brain activation connectivity and
00:46:53.180 then what I'm working on specifically is we have saliva, hair, and maybe blood forthcoming
00:47:00.580 hormones data and puberty measures and linking that to different brain and psychological outcomes
00:47:08.040 of interest. Wow. That's really interesting. You said that you said yesterday you had an issue
00:47:13.240 with functional MRIs and kids. So there are some issues that people have known about for a long
00:47:19.040 time one is just that kids move in the scanner and you have to lie really still so you can get
00:47:25.300 motion artifacts that just mess up your data but you know you can you can teach kids to stay still
00:47:31.200 and even if you have to throw out half the data if you get enough kids you're gonna have enough
00:47:36.080 usable data so that's more just a practical challenge but it can be overcome the other one
00:47:43.420 is if you're averaging across dozens or hundreds of brains in a study, you can get a really precise
00:47:51.820 estimate of, on average, what brain region is active during a particular task. So like I study
00:47:58.740 reward sensitivity, so often you'll use a gambling game paradigm, and basically you're measuring brain
00:48:03.840 activity during the whole game, and what you want to do ideally is filter out, say, wins versus
00:48:11.260 losses because if you're just looking at straight win activity you're not getting just reward
00:48:16.840 response in the brain you're also getting what is the visual cortex doing when you're looking at the
00:48:21.400 screen you're getting how is the motor cortex preparing to press the button because you have
00:48:25.740 to press the button for the gambling game but if you have win minus loss then in principle you
00:48:30.520 filter out all of the stuff that's the same in the task across whatever condition and you're
00:48:36.560 isolating, what is my brain doing uniquely when I win money compared to when I lose money?
00:48:42.360 And there, very robustly, you'll see a response in the ventral striatum, which is known to be
00:48:48.260 the brain region primarily responsible for reward processing. And especially in addiction models,
00:48:55.560 you'll see this. It's part of the mesolimbic mesocortical dopamine system. It's dopaminergic.
00:49:01.660 testosterone influences ventral striatal development and dopamine function. And all
00:49:08.040 that's very well established. The part that's trickier, and this is the part that's troubling
00:49:13.200 for my research, if you're looking at group averages, either in what part of the brain is
00:49:18.760 responsive, or how does it change with age on average, you can get very good estimates of that.
00:49:23.700 If you're trying to make individualized predictions, you can say, you know, on average,
00:49:28.900 does more testosterone correspond to more ventral striatum activity? And the answer is yes. But if
00:49:35.340 you want to make an individualized prediction, does more circulating hormone levels in your
00:49:40.480 blood correspond to more brain blood flow in this one participant? So the thing that would actually
00:49:46.200 be translational, the answer is no. And not only that, but sometimes that can be backwards. So
00:49:52.060 think about what the hormone levels that we're measuring. First of all, it's a single time point,
00:49:57.740 So it could be confounded, especially if you're measuring it through saliva, which most psychology research does because it's cheap and it's easy.
00:50:05.760 You know, you don't have to stick a needle in their arm, but it's really noisy.
00:50:09.380 It can be influenced by the last thing you've eaten, how much you slept, what your stress levels are, a whole host of confounds.
00:50:15.920 So ideally, you'd want a whole bunch of samples, maybe day to day, week to week, month to month, especially for females, because then you can also get menstrual cycle fluctuations messing with hormone levels.
00:50:28.920 So you want a lot of samples to be able to confidently assess the estimate.
00:50:35.180 But then even if you can do that, circulating hormone levels doesn't tell you what the individual genetic response to that is going to be.
00:50:45.920 So you could have someone with twice the testosterone as me, but functionally it's as if they have less because their androgen receptors aren't as sensitive or vice versa.
00:50:56.000 Can you test that at all or no?
00:50:57.780 Maybe.
00:50:58.320 That's a question for a physician.
00:51:00.120 I haven't, yeah, I haven't heard of anybody's ability to test androgen receptor sensitivity.
00:51:04.700 Maybe.
00:51:05.340 We could talk more about testosterone and reward sensitivity.
00:51:09.240 Yeah. 0.98
00:51:10.000 So this might be a really stupid question, but I don't know what I'm talking about here. 0.98
00:51:14.020 but you said there was a relationship there do men get like a higher dopamine release 0.99
00:51:21.360 in their reward system because of testosterone or is that not true is that just wrong you can tell
00:51:27.440 me i'm pausing because there's a version of the answer that's yes and a version of the answer
00:51:33.360 that's no okay so we do know that testosterone influences the structure and function of the
00:51:41.040 ventral striatum. So it's going to make it more reward sensitive. Now, does that mean that it's
00:51:46.160 literally more dopamine or does it just mean it's more sensitive? So this brings up an important
00:51:52.080 distinction in endocrinology research, organizational versus activational effects of hormones. So
00:51:57.520 organizational refers to these developmental effects. Anything physiological is developmental.
00:52:03.500 So the physical changes that happen during puberty for either sex, those are pretty much permanent.
00:52:08.760 this is why again for the puberty blockers debate it's such a big deal because if you are one of
00:52:14.260 these people with innate gender dysphoria and you do want to transition then early intervention is
00:52:21.000 necessary to block development of these opposite sex traits but there's also other questions about
00:52:28.080 can children consent and we can get back into that but organizational effects basically are
00:52:32.860 permanent so for the body those are more straightforward for the brain it has to do with
00:52:37.940 again, sensitive or critical windows of brain development. And then activational effects is
00:52:43.700 how is your brain and behavior changing in response to circulating hormone levels?
00:52:49.580 So take the testosterone away, the reward sensitivity goes away, the sex drive goes away.
00:52:55.080 And you see those studies both in experimental animal research where rodents may be castrated
00:53:01.460 or given hormone replacement, and you'll see these massive effects, or even in human clinical
00:53:06.780 populations like testicular cancer patients, if they have to be castrated, then you'll see sex
00:53:12.180 drive and motivation plummets. And then they go on testosterone replacement therapy, and it comes
00:53:16.960 back. And not only that, but they can go back on a fraction of once they had, and it's enough to
00:53:23.620 reinstate sex drive. So it's not the dose response relationship that most people think of. It's not
00:53:29.660 you have 10% more testosterone, so you're going to have 10% more aggression or sex drive, in part
00:53:35.820 because again circulating hormone levels doesn't tell you much about how it's actually bioavailable
00:53:41.740 but then beyond that it's not a dose-response relationship it seems to be more of this
00:53:47.140 threshold effect of you have enough to turn the sexual system on or you don't or same thing with
00:53:54.240 aggression status competition and if you have unnaturally high steroid levels then you can
00:54:00.000 also see increased sex drive or aggression but a little bit more testosterone isn't going to
00:54:05.520 correspond necessarily to a little bit more of those behaviors. That's interesting. So for women
00:54:10.280 and their reward system, do you think the amount of testosterone they have is enough to turn that 0.89
00:54:14.560 on? Is that how it works? Or is it just different in women? So here's what I think is an open
00:54:20.040 question. We know for sure that males are more reward sensitive and more prone to risk-taking,
00:54:26.480 especially during adolescence. So wait, can we pause? So what exactly is reward sensitive?
00:54:32.540 Reward sensitive, it could be actual response in the brain, how much ventral striatum activity
00:54:38.320 and how much dopamine release you see. It could also be in one of these gambling game studies,
00:54:43.580 like how willing are you to take a risk for potential reward? Or if you're talking about
00:54:49.440 sex drive reward, there have been interesting studies where you'll show participants porn
00:54:54.500 in the scanner and you can measure physiological arousal. You can measure skin conductance.
00:54:59.780 In men, you can even measure the pressure of their downstairs arousal.
00:55:07.540 And those are a bunch of individual differences that may or may not correlate with testosterone
00:55:13.820 levels.
00:55:14.360 But at the very least, if you completely eliminated it, like with androgen blockers, then that
00:55:19.680 would go away.
00:55:20.800 So there's some baseline level.
00:55:23.060 And what I'm not sure of, the open question here, is the female level the baseline?
00:55:27.780 and then what you do is you say males have more of this, therefore they're more likely to take
00:55:32.400 risks. But you also see that beginning at puberty, women are more anxious and more depressed, or at
00:55:39.780 least have a propensity for it. We don't know, is this because testosterone inhibits anxiety? And
00:55:46.680 there's some animal research to suggest that, and even anecdotal experience for people who take
00:55:51.520 testosterone replacement therapy. But it also could be that ovarian hormones, estrogen and
00:55:56.560 progesterone, increase anxiety and depression. And there are all sorts of evolutionary theories
00:56:00.980 for this. So it makes sense for males to be more reward sensitive and more willing to take risks
00:56:06.560 because one man can in principle impregnate dozens of women. So if he can take some major risk for
00:56:13.460 acquiring status or if he's more aggressive, as long as he can fertilize those eggs, even if he
00:56:19.120 gets himself killed shortly after, even if 90% of people who try get themselves killed, it can be a
00:56:25.620 stable reproductive strategy for men. And then it makes sense why women would try to be more 0.98
00:56:31.280 cautious, especially because women take care of children cross-culturally and have to be more
00:56:38.960 threat sensitive and protective of these vulnerable infants. You also see interesting interactions
00:56:44.980 with oxytocin here. They call it the mama bear effect. So oxytocin, it's known as the love
00:56:51.080 hormone, the cuddle hormone, but it can increase aggression. And what it really does is it
00:56:57.480 increases the strength of an overall in-group or out-group response. So it increases care towards
00:57:04.160 the in-group, but it also through a chemical cousin of it called vasopressin increases aggression
00:57:11.260 towards the out-group, this mama bear aggression. That's so interesting. I'm going to step away from
00:57:17.560 that a little bit. I want to talk about how has university been like for you in the political
00:57:24.960 landscape that we're in now? I know what it was like for me in 2012, and it was troublesome for
00:57:29.880 me then, but you're at Harvard. So what's it like there politically? Is it as crazy as it looks in
00:57:38.200 the news? Definitely not as crazy as in the news, but you do see some of those issues. So here's
00:57:44.680 the interesting thing. I think institutionally, there's really not as much of a problem with free
00:57:50.840 speech. Like you do see some examples of professors getting canceled, but even if they're canceled,
00:57:56.120 what it often looks like is you're disinvited to speak at a conference or something like that.
00:58:01.280 And that's terrible. That shouldn't happen. Like you should encourage disagreement and debate,
00:58:05.420 but very rarely are people losing their jobs, at least at Harvard. It does happen at places.
00:58:11.280 You know, even Carol Hoeven's story, which is the most extreme example that I know of at Harvard. She wasn't officially fired for going on Fox News and talking about the binary reality of sex and talking about her book on testosterone. But socially, people just made her life there such hell that she was essentially forced to leave.
00:58:33.020 So that's what you see more of. It's almost like bullying or passive aggressive. It's like this sense of false common knowledge, like in the Emperor's New Clothes story, this idea that everyone is thinking that this thing you can't say, even if most people themselves are thinking it.
00:58:53.960 And for whatever reason, maybe I'm more disagreeable. Maybe it's because I had a more atypical social upbringing. So I'm not as attuned or as willing to follow those sorts of social norms. Generally, I always speak my mind. And I've doubled down on this really ever since I started listening to your dad and exploring my faith more and believing in this foundational logos.
00:59:17.720 so now I think whatever the price is for saying what I believe to be true I'd much rather pay
00:59:24.780 that price than speak falsehood or hold my tongue so that has worked and it's it's very polarizing
00:59:33.380 I'll say that it's made the people who admire and respect me I think admire me and grow close to me
00:59:42.140 in that way a lot. And it's led to opportunities like this, like being on your podcast. But it's
00:59:48.360 also made a bunch of other people think, OK, that's the controversial guy that's interested
00:59:54.300 in evolutionary psychology and hormones and sex differences. And he's into Jordan Peterson and
01:00:00.240 just stay away. He's this bad conservative guy. And, you know, I'm also a diversity fellow at
01:00:08.260 Harvard. So this is on the DEI committee, which I applied for this role because I had this sense
01:00:15.080 that those committees, despite diversity being in the name, were not very diverse. So I actually
01:00:22.040 wrote my application basically saying this, like I have more conservative, unorthodox views. I
01:00:29.340 actually called myself conservative Christian and I was raised around Christianity. My take is
01:00:34.660 more abstract. We spoke about this a little bit. So I think it's very far from what that term
01:00:41.580 probably rings a bell is like MAGA evangelical Republican. And I'm quite far from that. But
01:00:48.360 my stance to DEI is even if I was that, there's no reason that shouldn't be welcome. That's
01:00:55.360 genuine diversity. That was your essay? Not in those terms. I didn't say even if I was a MAGA
01:01:02.320 republican i should be able to serve on this committee but you know that's that's the spirit
01:01:06.500 that inspired me to apply and the interesting thing you know i have mixed feelings about this
01:01:11.680 because on one hand i'm able to express my opinion and i'm never going to get in trouble for it
01:01:18.300 because you know it is expressing diversity i'm not making personal attacks i'm literally just
01:01:24.520 saying here's my opinion if you disagree i welcome that like tell me why you disagree like let's
01:01:30.940 debate I find that stuff fun I think some people find it intimidating or they worry are there going
01:01:37.120 to be social or career consequences to this and I think there are like I'm I'm playing a bit of a
01:01:42.420 gamble here I think it's going to work out for me like I have faith that it'll work out for me
01:01:46.800 but I don't know how it's going to be received crazy crazy okay okay this reminded me I thought
01:01:57.020 this was interesting because like I feel like most of my audience is older than university so
01:02:00.720 they don't know what's going on now. How has AI impacted people in university? Are they using it?
01:02:07.240 Are professors allowing it? Can they test for it? I think it's left up to each professor. I don't
01:02:14.820 think the university has an official stance on it. So some professors say it's completely banned,
01:02:21.200 but then I guess that means they do have to test for it somehow. I don't know how the tests go for
01:02:27.100 it. But I don't think most students are using it, at least not like it's going to write your essay
01:02:32.080 for you because it's really bad. It's really bad. It'll make up citations. It'll just say things
01:02:37.080 that are completely wrong. I found that it can be good for summarizing papers. But even if you do
01:02:43.720 that, it's like it depends what you want. If you want literally a one paragraph high level summary
01:02:49.360 and you don't need any nuance, then it's perfect for that. As soon as you need nuance, which you
01:02:55.180 need a lot of in grad school and in research then it's useless yeah okay okay interesting so let's
01:03:01.540 get back into into faith because you told me yesterday you've done an insane amount of research
01:03:06.920 on religions around the world and different philosophers so like where are you what what do
01:03:13.280 you believe right now so insane amount of research to me signifies like i'm doing a phd in it and my
01:03:19.340 gut instinct is this is just like a side personal interest so I couldn't tell you much about the
01:03:25.400 history of it but personally it stems from I think part growing up exposed to Christianity
01:03:35.020 but then thinking there was no substance to it like learning about evolution and not having
01:03:43.400 people I could go to and have critical discussions about it like people who say you know take it
01:03:48.520 literally, or leave it. So I decided to leave it. And it turned into actually this anti-theistic
01:03:53.960 phase in my teenage years. But in retrospect, I look at that like I was already depressed. I was
01:04:01.460 overweight and resentful and spending all my time online with this bunch of sickos. And, you know, 0.96
01:04:09.240 it's not surprising to me that I had such a pessimistic attitude towards faith. It's like
01:04:14.480 the Cain mindset, Cain and Abel. That resonated a lot. Your dad's Bible series resonated a lot
01:04:21.180 with me in college. And it was for two reasons. The first was because he took a very secular
01:04:28.040 approach to it. He's integrating it with evolutionary psychology rather than putting
01:04:32.580 it at odds with it. And secondly, he really talks about the depths of those negative experiences,
01:04:39.400 Like this resentful envy that one can experience. And I experienced this going to USC. You know, on one hand, I went on scholarship. So I applied through QuestBridge, the scholarship for low income students coming from a single mom. I wrote my essay about that.
01:04:58.220 I wrote about how growing up in poverty with a single mom, and I spent most of my time online, that I had to do things like learn how to shave, learn how to tie a tie from YouTube tutorials, and that it taught me this level of independence.
01:05:16.120 It gave me research skills and how I want to translate that into actual academic research.
01:05:21.320 So that worked out for me.
01:05:23.160 But then, you know, it's like a rich party school.
01:05:26.100 the make fun of it nickname for it is university of spoiled children and there were a lot of those
01:05:32.900 so i didn't fit in part of that was because of my own social difficulties but there's it's i would
01:05:39.580 oscillate between this resentful victim attitude like look at all these people who are coming from
01:05:45.080 wealthy two-parent households i don't fit in here they went to private school they're so much more
01:05:50.400 articulate than me. I don't know what I'm doing. And then simultaneously thinking, like, I'm going
01:05:56.160 to one of the best colleges for free. And other people across the world are starving or can't get
01:06:04.520 an education whatsoever. Like, what right do I have to be a victim? And I would go spiral in those
01:06:10.800 states. And I think it was just this inherent interest in science and philosophy. I couldn't
01:06:17.800 tell you why i had that so that's a cool thing too it's like it's almost like it was a protective
01:06:22.800 factor for me because i could have been out causing a bunch of trouble i did that online a
01:06:29.380 little bit like the scamming people on video games luring was this thing you'd do in runescape so
01:06:35.300 like there'd be pvp areas where players could fight each other and if you were a young kid like i was
01:06:41.100 when i first started playing people could trick you into coming there they'd kill you and take
01:06:44.760 their stuff. So then rather than learning not to do that, I learned to do it to other kids.
01:06:51.060 And then I would even justify it as in like, well, if you're that naive, you deserve to have
01:06:56.060 your stuff taken. And that's probably the same attitude that whatever older kid killed me 0.83
01:07:00.640 in my first couple of years playing the game did. So it's like I was morally grasping for something
01:07:08.280 right and trying to justify my own existence and maybe being more anxious and guilt prone
01:07:13.920 it wasn't just I'm going to get here and exploit as much as possible and then try to get a job
01:07:20.440 and make the most money as possible I think part of that was engaging with these philosophical
01:07:28.100 ideas pretty early on and it was through game theory that I started to really wrestle with
01:07:35.440 objective morality. It was through that, as well as this famous Gödel's incompleteness theorem.
01:07:41.220 And the basic idea there is that Gödel, famous mathematician, proved that any mathematical
01:07:49.180 system, you can make it logically coherent with a given set of axioms, and you can prove anything
01:07:56.360 within that structure, but you have to accept axioms. So that's almost like a leap of faith,
01:08:01.440 Right. And I started reflecting more. It was really after listening to the Bible series that even though I called myself such a secular, rational empiricist, like where did my idea that truth is good come from?
01:08:17.100 Where did my idea even that the universe has an organized structure come from?
01:08:22.040 Like, I realized that I had, at the very least, a metaphysics of physics.
01:08:27.160 Like, I believe there was this set of natural equations that could explain the world.
01:08:31.280 And I just took it as a given that that was there.
01:08:34.800 That's kind of a faith-based belief.
01:08:38.220 It's obviously evidence-based.
01:08:40.520 Like, you can prove that these laws are out there.
01:08:42.600 But then I started to ask, is the same thing true for morality?
01:08:46.620 and I think the conclusion that many of these secular thinkers come to is no like you can make
01:08:53.020 an evolutionary argument for it why cooperation is sometimes adaptive but then you'll come up with
01:08:57.780 all these different exceptions and cultural relativism to it but it was listening to this
01:09:05.000 bible series and particular the union psychology that was in it and connecting that to this idea
01:09:10.760 of archetypes, there was a line that your dad had about how Dawkins' memetic evolution,
01:09:18.260 that memes undergo natural selection, so like a joke can be modified to be made funnier and spread
01:09:23.500 more, or it can just die, that an archetype is something like the most extreme asymptotic
01:09:30.040 destination that a meme converges towards. So then there was this thought of there's that,
01:09:35.760 and then in game theory there are these equilibria these optimal solutions that you can reach and
01:09:42.160 some of them are cooperative and there was just this stitching together with that i also read
01:09:47.560 maps of meaning at this time and what did you what did you think of that it was did you understand
01:09:52.900 it it took me a while you know i was listening to the class lectures alongside that it's like
01:09:58.840 dense 500 pages but this was while i was taking the neuroscience so i was familiar with a lot of
01:10:04.520 the references and I was getting really into the philosophy. So it made like it literally changed
01:10:08.960 my life hands down. So that's why it's so cool to be doing this. Oh, that's cool. That's very cool.
01:10:15.600 I started definitely taking a more metaphysical orientation, but trying to string it together.
01:10:21.600 Like, okay, so morality is definitely reflective of our evolutionary psychology,
01:10:27.400 but it's not that it's just subjective and arbitrary there's going to be some almost
01:10:35.700 mathematical laws that guide us in a particular direction and that's going to be whatever this
01:10:41.500 optimal solution is if you're framing it in a game theory economic way like trying to maximize
01:10:46.620 cooperation or resources but then there's also the narrative organizing part that's talked about
01:10:52.940 in maps of meaning so this idea that information itself like memes can be operated on in terms of
01:11:02.900 natural selection and that those could co-evolve with humanity so that our moral intuition can
01:11:10.300 really co-evolve with these evolving morals that are transmitted in stories and then thinking okay
01:11:18.440 there are universal patterns in different world mythologies and world religions, and those are
01:11:24.300 reflective of our evolutionary psychology in some deep way. So you can still make the case, okay,
01:11:29.960 it's arbitrary, it's a reflection of the human psyche, but at the very least, it's not arbitrary.
01:11:36.220 It's like deep in our nature, and then it's, I think, our responsibility to figure out what
01:11:42.420 those stories are telling us. That was great. I feel like that made more sense to me than
01:11:48.400 dad explaining maps of meaning just so you know that was good that's a really high call yeah
01:11:53.740 that was really good wow okay so do puberty blockers if you give them to people when they're
01:12:01.820 like three and they take them throughout puberty can they ever recover from that like can you go
01:12:06.920 back to a normal brain structure after that do they have research on that okay so puberty blockers
01:12:12.940 were FDA approved in the United States in 1993. And they were approved for precocious puberty,
01:12:20.880 which is a medical diagnosis where you're beginning puberty way too early, like below age
01:12:26.780 eight or even below age seven. And they work, they're called GnRH agonists, gonadotropin
01:12:33.600 releasing hormone. This is a hormone that's produced in the hypothalamus that signals to
01:12:38.480 the pituitary gland to release other hormones that signal to the gonads testes in males to begin
01:12:44.920 producing testosterone or ovaries to produce estrogen in females and what they do at first
01:12:51.340 I thought that it was going to be an antagonist a blocker right but the cool thing the way the
01:12:56.060 hypothalamus releases GNRH it works in a pulsatile fashion so it goes up and down and your brain is
01:13:02.060 expecting a certain rhythm and the agonist it just floods it it's like constant activation
01:13:07.280 we actually don't know whether it's is it that it becomes desensitized so then your brain just
01:13:14.720 no longer responds or is it just the fact that it's constant activation so it's not following
01:13:19.140 the rhythmic pattern so then it doesn't work because your brain only responds to that rhythm
01:13:23.340 we don't know but either way it's an agonist that essentially shuts off the remaining cascade
01:13:28.940 and it specifically inhibits gonadal pubertal development so there's adrenal driven pubertal
01:13:37.920 development so this would be like the sex non-specific secondary sex characteristics
01:13:41.960 like body hair acne body odor that stuff still happens if you're given puberty blockers so that's
01:13:49.320 perfect for someone with precocious puberty so you're getting you're gonna still go through
01:13:54.080 the early pre-pubertal development, but you're not going to be growing secondary sex characteristics
01:14:02.260 so early, like at age six or seven. So the drugs work very well for that. And then the theory for
01:14:11.000 youth with gender dysphoria was going to be, well, you're already experiencing gender dysphoria as
01:14:18.140 a result of your birth assigned sex. And magnifying those characteristics is only going to make it
01:14:24.640 worse. So the theory here is we block that and it'll at the very least give you more time to
01:14:31.740 think, so they say, and decide whether you want to pursue puberty, whether the gender dysphoria
01:14:38.500 resolves or not, or whether you want to transition. And over 90% of kids who are prescribed
01:14:45.880 puberty blockers. So if they're prescribed, it's off label. It's not illegal, but it was originally
01:14:51.220 meant and approved for precocious puberty. And it was more theoretical proposed that, well,
01:14:56.820 it's very intuitive, right? Like if you experience gender dysphoria, you want to be the opposite sex. 0.92
01:15:02.720 These hormones are the things that give you the opposite sex traits, then very well, it might 0.98
01:15:08.280 reduce your symptoms. But also you could imagine a comparison to anorexia. Like if you're experiencing 0.62
01:15:14.760 extreme body dysmorphia and you think you're overweight, then maybe weight loss pills would
01:15:20.160 make your symptoms go down. But with anorexia, it generally just spirals and it can become
01:15:26.620 life-threatening. So for me, that's a really open question. And I think people need to be
01:15:32.020 talking about it more. I'm not opposed to the idea that gender dysphoria would resolve. And
01:15:37.440 I think that if it's, say, the innate case, like what you see with 5-alpha reductase,
01:15:43.880 where it could go either way, beginning at puberty, you start to experience these masculinizing
01:15:49.900 features. And maybe you decide you want to live your life as a man, even though you didn't know
01:15:54.140 you were one, or maybe you decide you want to block them and continue as a woman. I think 0.96
01:15:59.000 for those types of people, it's probably the right decision. But you also see in recent years,
01:16:05.920 the rate of gender dysphoria cases has skyrocketed like more than 10 times the amount of previous
01:16:14.980 decades and also the sex ratio has swapped so historically yeah it's been more males who
01:16:21.800 transition to be woman and now with gen z girls it's mostly females identifying as either non-binary
01:16:31.120 or trans men, and they're taking puberty blockers. So again, puberty blockers, if they are harmless 0.97
01:16:39.520 when it comes to physiological and reproductive development, with one exception, the one exception
01:16:45.680 is bone density. And that's because sex steroids fuse the growth plates. So the adrenal processes 0.95
01:16:52.800 that facilitate height, growth spurt, those are separate hormones. If you block GnRH,
01:16:59.480 that pathway still continues. So this, you can see in people who were castrated in eunuchs,
01:17:07.840 there are historical records going back centuries that they often grow to be really tall and lanky
01:17:13.000 because again, they have excess growth hormone and there's no gonadal hormones that fuse the
01:17:18.280 growth plate and inhibit that growth hormone signaling. Oh wow, I had no idea about that.
01:17:22.780 right so bone density can be compromised but as long as you have the sex steroids later that will
01:17:31.420 resolve and reproductive function again as long as you go through puberty your birth sex puberty
01:17:38.480 i don't think there will be any long-term harms to reproduction but two problems there the first
01:17:45.700 is most of them do go on cross sex hormones and those are more likely to impair fertility
01:17:51.520 And then second, well, the only thing we know is that adolescence is a sensitive period
01:17:56.100 for brain development that's expectant of certain hormonal inputs, and that the hormones
01:18:00.460 are involved in these high-level cognitive processes, like reward sensitivity, like social
01:18:05.420 cognition, like emotion regulation, and that they're somehow related to these massive sex
01:18:09.740 differences that we see that have health implications, like risk-taking in males or anxiety and depression
01:18:15.040 in females, but at the very least, we know that they're going to be influenced, and it
01:18:19.160 could be for the better but just this theory that you're making unpredictable changes and we don't
01:18:26.140 even understand on the individual differences level how it's going to impact anxiety or depression
01:18:30.120 and we do know from limited animal studies that at the very least it impacts cortical development
01:18:36.140 structurally we don't know exactly what the function would be there's limited evidence
01:18:40.380 that's been contested that it might increase anxiety-like behaviors or depressive-like
01:18:45.320 behaviors which would be really ironic and unfortunate because the whole point of it is
01:18:50.000 to decrease these internalizing symptoms yeah okay so there was just this massive review the
01:18:57.900 cass review oh yeah billary cass it was funded by england's nhs their nih equivalent and really it
01:19:06.840 it just said that we don't have any high quality longitudinal evidence we can't make any claims
01:19:12.980 whether these things are good or bad. The theory, again, is intuitive. Maybe gender dysphoria will
01:19:18.700 resolve, but there's all these open questions about why are so many kids, so many more kids
01:19:24.200 than before coming out as trans? It could be because we're more accepting, there's less stigma.
01:19:29.800 I think that could explain a small part of it, but probably not this massive spike that
01:19:34.880 seems limited just to Gen Z, and it doesn't explain the sex ratio swap. So that's talked
01:19:41.020 about in the review. And then the review gestures at their unknown effects on brain development.
01:19:50.500 And no one's studying that in humans. No, that's not true. There are a few clinical trials that
01:19:56.040 are being done. We still don't have the data. And what you really want is longitudinal evidence.
01:20:01.640 There are also some animal studies. But the open question and the recommendation from the
01:20:08.180 CAS review is, we shouldn't call this treatment before we know if the benefits outweigh the harms,
01:20:13.140 even though there might be potential benefits, especially for these types of intersex conditions
01:20:19.660 where due to genetic abnormalities or due to hormonal imbalances, you might stand very well
01:20:25.260 to benefit from it. But given the unknowns, especially for brain development in typically
01:20:30.540 developing children, this shouldn't be prescribed outside of clinical trials. It's a separate
01:20:36.600 question, like an ethical question, whether to even do the clinical trials, whether children
01:20:41.160 can consent to them. I don't know if we have time for all that. And plus that's more subjective. So
01:20:46.280 I'll leave it at that. That's where the evidence stands. Okay. That's not, that's like the nerdiest
01:20:51.020 way of saying this was a bad idea that I've ever heard, but okay. Okay. So then delving into that
01:20:57.400 a little bit, doesn't it seem reasonable because there are so many more girls that are interested
01:21:02.900 or or going towards or gender dysphoric or that it's kind of 2024's version of hysteria
01:21:09.700 like aren't women more impacted by these kind of social disorders like i know anorexia if you're
01:21:15.860 friends with a bunch of people that have anorexia then you're more likely to get that right
01:21:19.020 there's really good social network analysis on this so yeah anorexia body image disorders
01:21:24.640 cutting suicidality smoking in both sexes but those former cases especially in girls you
01:21:31.380 definitely see peer influence. Hysteria, I know that there were those historical cases. I don't
01:21:38.000 know enough about those, but the natural parallel to me. So the other thing I'll say, this is such
01:21:44.320 a polarized topic, but I have a pretty optimistic take on it. The optimism is that you can tell that
01:21:51.080 everyone wants the well-being of the children. Top priority, right? You just have very different
01:21:56.940 ideas of what that looks like. For some people, they think the hormones are life-saving and it's
01:22:02.180 what the children need. For other people, that hormones might destroy the child's life. They
01:22:06.980 can't consent. They want to protect the children. So the good news is everyone wants to protect the
01:22:11.000 children, right? Like we really care about this and it's really important research and we need
01:22:16.080 high quality longitudinal evidence. So that's being done. That's a good thing. I also think
01:22:20.900 there's this lingering collective conscience about how we used to treat the same lbgt populations 0.99
01:22:29.620 so homosexuality used to be in the dsm as a mental disorder not only that they used to give hormonal 0.77
01:22:37.640 treatments for it it was really chemical castration you know the story of alan turing 0.86
01:22:42.520 let's go through it father of modern computer science he helped crack the germans cryptography
01:22:51.240 code during world war ii helped win or end it early and built the first modern computer in 0.61
01:22:59.700 order to do that he was also gay and he was closeted this whole time while in the uk it was 0.68
01:23:06.820 a criminal offense. And he had all these top secret security clearances. Once it was outed
01:23:13.880 that he was having these secret affairs with men, rather than put him in jail, he was given this
01:23:19.780 option to go on chemical castration. And it made him severely depressed. I don't know what he was
01:23:26.860 given exactly. It may have been testosterone blockers. It probably wasn't just that. So it
01:23:31.940 eliminates sex drive, but it also eliminates motivation. It seems that it made him severely
01:23:38.280 depressed and he killed himself. So we lost a great mind because of that. And conversion therapy
01:23:44.700 was a disaster. So the fundamental question for transgender identity is, is it more similar to 0.96
01:23:50.620 sexual orientation born this way? Or is it more similar to anorexia, social contagion? I think
01:23:57.600 the answer is both for different people. That's why I really wanted to talk about this famous
01:24:02.000 twin study and the intersex conditions, because I do believe firmly that there's an innate
01:24:07.640 biological cause, at least in some people, probably in earlier cohorts in most of them, 0.89
01:24:12.760 that gives them gender dysphoria, that makes them really risk their life to go through this
01:24:18.720 transition back when the stigma was way, way higher. It's still a problem. But the fact that
01:24:24.240 people used to do it decades ago, and that it used to be mostly males who experienced that,
01:24:30.000 that's an interesting research question to me. And I think something about the increased rates
01:24:35.260 combined with the sex ratio swap that makes me think it's more likely that in probably the
01:24:42.360 majority of Gen Z trans men, females identifying as men, that that is probably not the same
01:24:53.560 biological innate cause. It's probably manifesting late onset during puberty. So there's this term
01:25:02.220 late onset or rapid onset gender dysphoria. And this is the social contagion hypothesis.
01:25:08.480 It's gotten a whole lot of flack. And to be clear, there's no data to support it.
01:25:15.200 I think it's a very interesting hypothesis. And I don't think if it's true, that it's true for
01:25:22.360 everyone. That's this binary thinking that I think is the root of this problem. It's not going to be
01:25:27.580 a one-size-fits-all solution. Interesting. Interesting. I'm probably in the more of the
01:25:35.160 camp that maybe 1% of the people you see now that are experiencing gender dysphoria
01:25:42.020 actually have a biological root and a real problem. I feel like the rest of it might 0.98
01:25:48.280 be a social contagion right that's but like i have no data for that that's just what it looks
01:25:53.540 like yeah i think so it's it's all a real problem and it's all going to have serious psychological
01:25:59.940 side effects that everyone wants to treat whether that's through therapy whether it's wait watchful 0.68
01:26:05.620 waiting and its children are going to grow out of it maybe they're going to turn out to be gay
01:26:09.740 and otherwise completely healthy cisgender i think that's probably the case for some again for some
01:26:16.720 they might benefit from puberty blockers or hormone replacement. But again, we have no evidence
01:26:22.480 that that actually reduces negative mental health side effects. And there's theoretical reason that
01:26:29.680 maybe it would actually exacerbate them. But again, we don't have solid evidence of that either.
01:26:35.200 Yeah. Okay. That makes sense. Well, I'm officially mind blown. That was great.
01:26:42.240 Thank you. Yeah. Thanks very much for coming on.